日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Mouse models of neutropenia reveal progenitor-stage-specific defects

中性粒细胞减少症小鼠模型揭示祖细胞阶段特异性缺陷

David E Muench, Andre Olsson, Kyle Ferchen, Giang Pham, Rachel A Serafin, Somchai Chutipongtanate, Pankaj Dwivedi, Baobao Song, Stuart Hay, Kashish Chetal, Lisa R Trump-Durbin, Jayati Mookerjee-Basu, Kejian Zhang, Jennifer C Yu, Carolyn Lutzko, Kasiani C Myers, Kristopher L Nazor, Kenneth D Greis, D

Time resolved quantitative phospho-tyrosine analysis reveals Bruton's Tyrosine kinase mediated signaling downstream of the mutated granulocyte-colony stimulating factor receptors

时间分辨定量磷酸酪氨酸分析揭示了布鲁顿酪氨酸激酶介导的突变粒细胞集落刺激因子受体下游的信号传导

Pankaj Dwivedi, David E Muench, Michael Wagner, Mohammad Azam, H Leighton Grimes, Kenneth D Greis

Phospho serine and threonine analysis of normal and mutated granulocyte colony stimulating factor receptors

正常和突变的粒细胞集落刺激因子受体的磷酸丝氨酸和苏氨酸分析

Pankaj Dwivedi, David E Muench, Michael Wagner, Mohammad Azam, H Leighton Grimes, Kenneth D Greis

Clonal hematopoiesis of indeterminate potential and its impact on patient trajectories after stem cell transplantation

不确定潜力的克隆造血及其对干细胞移植后患者轨迹的影响

Derek S Park, Afua A Akuffo, David E Muench, H Leighton Grimes, Pearlie K Epling-Burnette, Philip K Maini, Alexander R A Anderson, Michael B Bonsall

miR-196b target screen reveals mechanisms maintaining leukemia stemness with therapeutic potential

miR-196b靶点筛选揭示了维持白血病干细胞特性的机制及其治疗潜力

Sara E Meyer ,David E Muench ,Andrew M Rogers ,Tess J Newkold ,Emily Orr ,Eric O'Brien ,John P Perentesis ,John G Doench ,Ashish Lal ,Patrick J Morris ,Craig J Thomas ,Judy Lieberman ,Edwina McGlinn ,Bruce J Aronow ,Nathan Salomonis ,H Leighton Grimes

Granulocyte-Monocyte Progenitors and Monocyte-Dendritic Cell Progenitors Independently Produce Functionally Distinct Monocytes

粒细胞-单核细胞祖细胞和单核细胞-树突状细胞祖细胞独立产生功能不同的单核细胞

Alberto Yáñez ,Simon G Coetzee ,Andre Olsson ,David E Muench ,Benjamin P Berman ,Dennis J Hazelett ,Nathan Salomonis ,H Leighton Grimes ,Helen S Goodridge

DNMT3A Haploinsufficiency Transforms FLT3ITD Myeloproliferative Disease into a Rapid, Spontaneous, and Fully Penetrant Acute Myeloid Leukemia

DNMT3A 单倍体不足将 FLT3ITD 骨髓增生性疾病转变为快速、自发且完全渗透的急性髓系白血病

Sara E Meyer, Tingting Qin, David E Muench, Kohei Masuda, Meenakshi Venkatasubramanian, Emily Orr, Lauren Suarez, Steven D Gore, Ruud Delwel, Elisabeth Paietta, Martin S Tallman, Hugo Fernandez, Ari Melnick, Michelle M Le Beau, Scott Kogan, Nathan Salomonis, Maria E Figueroa, H Leighton Grimes4