日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Monophosphoryl lipid A pretreatment suppresses sepsis- and LPS-induced proinflammatory cytokine production in the medullary thick ascending limb

单磷酰脂质 A 预处理可抑制脓毒症和 LPS 诱导的髓质粗升支促炎细胞因子产生

Bruns A Watts 3rd, Esther Tamayo, Edward R Sherwood, David W Good

Monophosphoryl lipid A induces protection against LPS in medullary thick ascending limb through induction of Tollip and negative regulation of IRAK-1

单磷酰脂质 A 通过诱导 Tollip 和负向调节 IRAK-1 来诱导延髓厚升支对 LPS 的保护作用

Bruns A Watts 3rd, Esther Tamayo, Edward R Sherwood, David W Good

Monophosphoryl lipid A prevents impairment of medullary thick ascending limb [Formula: see text] absorption and improves plasma [Formula: see text] concentration in septic mice

单磷酰脂质 A 可防止髓质粗升支 [公式:见文本] 吸收受损并改善脓毒症小鼠的血浆 [公式:见文本] 浓度

Bruns A Watts, Thampi George, Edward R Sherwood, David W Good

High-mobility group box 1 inhibits HCO3- absorption in the medullary thick ascending limb through RAGE-Rho-ROCK-mediated inhibition of basolateral Na+/H+ exchange

高迁移率族蛋白 B1 通过 RAGE-Rho-ROCK 介导的基底侧 Na+/H+ 交换抑制髓质厚升支中的 HCO3- 吸收

Bruns A Watts 3rd, Thampi George, Andrew Badalamenti, David W Good

A two-hit mechanism for sepsis-induced impairment of renal tubule function

脓毒症引起肾小管功能损害的双重打击机制

Bruns A Watts 3rd, Thampi George, Edward R Sherwood, David W Good

Toll-like receptor 2 is required for LPS-induced Toll-like receptor 4 signaling and inhibition of ion transport in renal thick ascending limb

Toll 样受体 2 是 LPS 诱导的 Toll 样受体 4 信号传导和肾脏粗升支离子转运抑制所必需的

David W Good, Thampi George, Bruns A Watts 3rd

Lipopolysaccharide directly alters renal tubule transport through distinct TLR4-dependent pathways in basolateral and apical membranes

脂多糖通过基底膜和顶端膜中不同的 TLR4 依赖途径直接改变肾小管运输

David W Good, Thampi George, Bruns A Watts 3rd