日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Glioma-derived CCL2 and CCL7 mediate migration of immune suppressive CCR2+/CX3CR1+ M-MDSCs into the tumor microenvironment in a redundant manner

胶质瘤来源的CCL2和CCL7以冗余的方式介导免疫抑制性CCR2+/CX3CR1+ M-MDSC向肿瘤微环境的迁移。

Gregory P Takacs ,Christian J Kreiger ,Defang Luo ,Guimei Tian ,Julia S Garcia ,Loic P Deleyrolle ,Duane A Mitchell ,Jeffrey K Harrison

CCR2 inhibition reduces tumor myeloid cells and unmasks a checkpoint inhibitor effect to slow progression of resistant murine gliomas

CCR2 抑制可减少肿瘤髓系细胞并揭示检查点抑制剂的作用,从而减缓耐药性鼠类神经胶质瘤的进展

Joseph A Flores-Toro, Defang Luo, Adithya Gopinath, Matthew R Sarkisian, James J Campbell, Israel F Charo, Rajinder Singh, Thomas J Schall, Meenal Datta, Rakesh K Jain, Duane A Mitchell, Jeffrey K Harrison

Angiotensin II Regulation of Proliferation, Differentiation, and Engraftment of Hematopoietic Stem Cells

血管紧张素 II 对造血干细胞增殖、分化和植入的调节

Seungbum Kim, Michael Zingler, Jeffrey K Harrison, Edward W Scott, Christopher R Cogle, Defang Luo, Mohan K Raizada

Expression and functional heterogeneity of chemokine receptors CXCR4 and CXCR7 in primary patient-derived glioblastoma cells

趋化因子受体 CXCR4 和 CXCR7 在患者原代胶质母细胞瘤细胞中的表达和功能异质性

Che Liu, Kien Pham, Defang Luo, Brent A Reynolds, Parvinder Hothi, Gregory Foltz, Jeffrey K Harrison

Hypoxia-induced endothelial CX3CL1 triggers lung smooth muscle cell phenotypic switching and proliferative expansion

缺氧诱导的内皮 CX3CL1 引发肺平滑肌细胞表型转换和增殖扩增

Jianliang Zhang, Hanbo Hu, Nadia L Palma, Jeffrey K Harrison, Kamal K Mubarak, Robin D Carrie, Hassan Alnuaimat, Xiaoqiang Shen, Defang Luo, Jawaharlal M Patel

Chemokine receptor CXCR3 promotes growth of glioma

趋化因子受体 CXCR3 促进胶质瘤生长

Che Liu, Defang Luo, Brent A Reynolds, Geeta Meher, Alan R Katritzky, Bao Lu, Craig J Gerard, Cyrus P Bhadha, Jeffrey K Harrison

Hematopoietic- and neurologic-expressed sequence 1 (Hn1) depletion in B16.F10 melanoma cells promotes a differentiated phenotype that includes increased melanogenesis and cell cycle arrest

B16.F10 黑色素瘤细胞中的造血和神经表达序列 1 (Hn1) 耗竭会促进分化表型,包括黑色素生成增加和细胞周期停滞

Katharine M Laughlin, Defang Luo, Che Liu, Gerry Shaw, Kenneth H Warrington Jr, Brian K Law, Jeffrey K Harrison