Deng Pan-Yue相关文献与解析，生知库 | 链接生命科学的每一步

Stunault Marion I, Deng Pan-Yue, Yadav Anjali, Periandri Erica M, de Luna Vitorino Francisca N, Thomsen Michael B, Sponagel Jasmin, Barfield Amelia J, Ponce Renzelle J, Foroughi Layla, Garcia Benjamin A, Egervari Gabor, Klyachko Vitaly A, Ashrafi Ghazaleh

神经科学

发表日期：2026

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Satellite Glial Cells Control Sensory Neuron Excitability via the Release of Fibulin-2.

卫星胶质细胞通过释放纤连蛋白-2来控制感觉神经元的兴奋性。

期刊:

影响因子:

doi:10.64898/2026.02.13.705760

Ansari Irshad, Deng Pan-Yue, Rosen Sarah F, Thomsen Michael B, Klyachko Vitaly A, Cavalli Valeria

Circuit-based intervention corrects excessive dentate gyrus output in the Fragile X mouse model.

电路干预可纠正脆性X染色体小鼠模型中齿状回过度输出

期刊:

影响因子:

doi:10.1101/2023.09.27.559792

Deng Pan-Yue, Kumar Ajeet, Cavalli Valeria, Klyachko Vitaly A

Mouse

发表日期：2023

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Channelopathies in fragile X syndrome

脆性X综合征中的离子通道病

期刊:Nature Reviews Neuroscience

影响因子:26.7

doi:10.1038/s41583-021-00445-9

Deng, Pan-Yue; Klyachko, Vitaly A

发表日期：2021

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Hyperexcitability of Sensory Neurons in Fragile X Mouse Model

脆性X染色体小鼠模型中感觉神经元的过度兴奋

期刊:Frontiers in Molecular Neuroscience

影响因子:3.8

doi:10.3389/fnmol.2021.796053

Deng, Pan-Yue; Avraham, Oshri; Cavalli, Valeria; Klyachko, Vitaly A

Genetic upregulation of BK channel activity normalizes multiple synaptic and circuit defects in a mouse model of fragile X syndrome

在脆性X综合征小鼠模型中，BK通道活性的基因上调可使多种突触和回路缺陷恢复正常。

期刊:Journal of Physiology-London

影响因子:4.4

doi:10.1113/JP271031

Deng, Pan-Yue; Klyachko, Vitaly A

神经科学

发表日期：2016

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Independent role for presynaptic FMRP revealed by an FMR1 missense mutation associated with intellectual disability and seizures

FMR1错义突变与智力障碍和癫痫相关，揭示了突触前FMRP的独立作用

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:9.1

doi:10.1073/pnas.1423094112

Myrick, Leila K; Deng, Pan-Yue; Hashimoto, Hideharu; Oh, Young Mi; Cho, Yongcheol; Poidevin, Mickael J; Suhl, Joshua A; Visootsak, Jeannie; Cavalli, Valeria; Jin, Peng; Cheng, Xiaodong; Warren, Stephen T; Klyachko, Vitaly A

The diverse functions of short-term plasticity components in synaptic computations

短期可塑性成分在突触计算中的多种功能

期刊:Communicative and Integrative Biology

影响因子:

doi:10.4161/cib.15870

Deng, Pan-Yue; Klyachko, Vitaly A

神经科学

发表日期：2011

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Modulation of GABAergic transmission by muscarinic receptors in the entorhinal cortex of juvenile rats

幼鼠内嗅皮层毒蕈碱受体对GABA能传递的调节

期刊:Journal of Neurophysiology

影响因子:2.1

doi:10.1152/jn.00226.2009

Xiao, Zhaoyang; Deng, Pan-Yue; Yang, Chuanxiu; Lei, Saobo

信号转导

发表日期：2009

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Serotonin increases GABA release in rat entorhinal cortex by inhibiting interneuron TASK-3 K+ channels

血清素通过抑制中间神经元 TASK-3 K+ 通道增加大鼠内嗅皮层中 GABA 的释放。

期刊:Molecular and Cellular Neuroscience

影响因子:2.4

doi:10.1016/j.mcn.2008.07.005

Deng, Pan-Yue; Lei, Saobo

Ketogenic diet dampens excitatory neurotransmission by shrinking synaptic vesicle pools.

生酮饮食通过缩小突触小泡池来抑制兴奋性神经传递。

Satellite Glial Cells Control Sensory Neuron Excitability via the Release of Fibulin-2.

卫星胶质细胞通过释放纤连蛋白-2来控制感觉神经元的兴奋性。

Circuit-based intervention corrects excessive dentate gyrus output in the Fragile X mouse model.

电路干预可纠正脆性X染色体小鼠模型中齿状回过度输出

Channelopathies in fragile X syndrome

脆性X综合征中的离子通道病

Hyperexcitability of Sensory Neurons in Fragile X Mouse Model

脆性X染色体小鼠模型中感觉神经元的过度兴奋

Genetic upregulation of BK channel activity normalizes multiple synaptic and circuit defects in a mouse model of fragile X syndrome

在脆性X综合征小鼠模型中，BK通道活性的基因上调可使多种突触和回路缺陷恢复正常。

Independent role for presynaptic FMRP revealed by an FMR1 missense mutation associated with intellectual disability and seizures

FMR1错义突变与智力障碍和癫痫相关，揭示了突触前FMRP的独立作用

The diverse functions of short-term plasticity components in synaptic computations

短期可塑性成分在突触计算中的多种功能

Modulation of GABAergic transmission by muscarinic receptors in the entorhinal cortex of juvenile rats

幼鼠内嗅皮层毒蕈碱受体对GABA能传递的调节

Serotonin increases GABA release in rat entorhinal cortex by inhibiting interneuron TASK-3 K+ channels

血清素通过抑制中间神经元 TASK-3 K+ 通道增加大鼠内嗅皮层中 GABA 的释放。