Devram S相关文献与解析，生知库 | 链接生命科学的每一步

日期影响因子

PPARγ mediated enhanced lipid biogenesis fuels Mycobacterium tuberculosis growth in a drug-tolerant hepatocyte environment

PPARγ介导的脂质生物合成增强促进结核分枝杆菌在耐药肝细胞环境中的生长

影响因子:6.4

doi:10.7554/eLife.103817.

Binayak Sarkar,Jyotsna Singh,Mohit Yadav,Priya Sharma,Raman Deep Sharma,Shweta Singh,Aakash Chandramouli,Kritee Mehdiratta,Ashwani Kumar,Siddhesh S Kamat,Devram S Ghorpade,Debasisa Mohanty,Dhiraj Kumar,Rajesh S Gokhale

细胞生物学

发表日期：2025

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Development and Validation of AAV-Mediated Liver, Liver-VAT, and Liver-Brain SORT and Therapeutic Regulation of FASN in Hepatic De Novo Lipogenesis.

AAV介导的肝脏、肝脏-VAT和肝脏-脑SORT的开发和验证以及FASN在肝脏从头脂肪生成中的治疗性调控

影响因子:5.2

doi:10.3390/cells14050372

Bhadury Ratulananda, Athar Mohammad, Mishra Pooja, Gogoi Chayanika, Sharma Shubham, Ghorpade Devram S

发表日期：2025

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Targeting Soluble DPP-4 for Insulin Resistance: Origin Matters

靶向可溶性 DPP-4 治疗胰岛素抵抗：来源至关重要

期刊:Journal of Clinical Endocrinology & Metabolism

影响因子:5.1

doi:10.1210/clinem/dgaa902

Ozcan, Lale; Ghorpade, Devram S; Tabas, Ira

免疫/内分泌

发表日期：2021

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Hepatocyte-secreted DPP4 in obesity promotes adipose inflammation and insulin resistance

肥胖患者肝细胞分泌的 DPP4 促进脂肪炎症和胰岛素抵抗

影响因子:50.5

doi:10.1038/nature26138

Devram S Ghorpade, Lale Ozcan, Ze Zheng, Sarah M Nicoloro, Yuefei Shen, Emily Chen, Matthias Blüher, Michael P Czech, Ira Tabas

细胞生物学

发表日期：2018

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Hepatocyte DACH1 Is Increased in Obesity via Nuclear Exclusion of HDAC4 and Promotes Hepatic Insulin Resistance

肥胖状态下，肝细胞DACH1表达增加，其机制是通过HDAC4核外排阻促进肝脏胰岛素抵抗。

期刊:Cell Reports

影响因子:6.9

doi:10.1016/j.celrep.2016.05.006

Ozcan, Lale; Ghorpade, Devram S; Zheng, Ze; de Souza, Jane Cristina; Chen, Ke; Bessler, Marc; Bagloo, Melissa; Schrope, Beth; Pestell, Richard; Tabas, Ira

细胞生物学

免疫/内分泌

发表日期：2016

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