日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

n-6 High Fat Diet Induces Gut Microbiome Dysbiosis and Colonic Inflammation

n-6高脂肪饮食诱发肠道菌群失调和结肠炎症

Selmin, Ornella I; Papoutsis, Andreas J; Hazan, Sabine; Smith, Christopher; Greenfield, Nick; Donovan, Micah G; Wren, Spencer N; Doetschman, Thomas C; Snider, Justin M; Snider, Ashley J; Chow, Sherry H-H; Romagnolo, Donato F

Interleukin-30 (IL27p28) alleviates experimental sepsis by modulating cytokine profile in NKT cells

白细胞介素-30 (IL27p28) 通过调节 NKT 细胞中的细胞因子谱来缓解实验性脓毒症

Yan, Jun; Mitra, Abhisek; Hu, Jiemiao; Cutrera, Jeffery J; Xia, Xueqing; Doetschman, Thomas; Gagea, Mihai; Mishra, Lopa; Li, Shulin

Cardiac-specific inducible and conditional gene targeting in mice

小鼠心脏特异性诱导和条件性基因靶向

Doetschman, Thomas; Azhar, Mohamad

Transforming growth factor beta signaling in adult cardiovascular diseases and repair

转化生长因子β信号通路在成人心血管疾病和修复中的作用

Doetschman, Thomas; Barnett, Joey V; Runyan, Raymond B; Camenisch, Todd D; Heimark, Ronald L; Granzier, Henk L; Conway, Simon J; Azhar, Mohamad

Fibroblast growth factor-2-induced cardioprotection against myocardial infarction occurs via the interplay between nitric oxide, protein kinase signaling, and ATP-sensitive potassium channels

成纤维细胞生长因子-2诱导的抗心肌梗死心脏保护作用是通过一氧化氮、蛋白激酶信号通路和ATP敏感性钾通道之间的相互作用实现的。

Manning, Janet R; Carpenter, Gregory; Porter, Darius R; House, Stacey L; Pietras, Daniel A; Doetschman, Thomas; Schultz, Jo el J

GI GEMs: genetically engineered mouse models of gastrointestinal disease

GI GEMs:基因工程改造的胃肠道疾病小鼠模型

Doetschman, Thomas

The inter-relationship of periostin, TGF beta, and BMP in heart valve development and valvular heart diseases

骨膜蛋白、TGF-β和BMP在心脏瓣膜发育和瓣膜性心脏病中的相互关系

Conway, Simon J; Doetschman, Thomas; Azhar, Mohamad

The influence of FGF2 high molecular weight (HMW) isoforms in the development of cardiac ischemia-reperfusion injury

FGF2高分子量(HMW)亚型在心肌缺血再灌注损伤发展中的影响

Liao, Siyun; Bodmer, Janet R; Azhar, Mohamad; Newman, Gilbert; Coffin, J Douglas; Doetschman, Thomas; Schultz, Jo El J

Disruption of the Fgf2 gene activates the adipogenic and suppresses the osteogenic program in mesenchymal marrow stromal stem cells

Fgf2基因的破坏会激活间充质骨髓基质干细胞的脂肪生成程序并抑制其成骨程序。

Xiao, Liping; Sobue, Takanori; Esliger, Alycia; Kronenberg, Mark S; Coffin, J Douglas; Doetschman, Thomas; Hurley, Marja M

Origin of cardiac fibroblasts and the role of periostin

心脏成纤维细胞的起源及骨膜蛋白的作用

Snider, Paige; Standley, Kara N; Wang, Jian; Azhar, Mohamad; Doetschman, Thomas; Conway, Simon J