日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

NSD2 targeting reverses plasticity and drug resistance in prostate cancer

NSD2靶向治疗可逆转前列腺癌的可塑性和耐药性

Li, Jia J; Vasciaveo, Alessandro; Karagiannis, Dimitris; Sun, Zhen; Gretarsson, Kristjan H; Chen, Xiao; Ouerfelli, Ouathek; Socciarelli, Fabio; Frankenstein, Ziv; Dong, Hanyang; Zou, Min; Yuan, Wei; Yang, Guangli; Aizenman, Gabriel M; Pannellini, Tania; Xu, Xinjing; Beltran, Himisha; Chen, Yu; Gardner, Kevin; Robinson, Brian D; de Bono, Johann; Gozani, Or; Abate-Shen, Cory; Rubin, Mark A; Loda, Massimo; Sawyers, Charles L; Califano, Andrea; Lu, Chao; Shen, Michael M

d-lactate drives lysine d-lactylation to regulate metabolism in Escherichia coli

d-乳酸驱动赖氨酸d-乳酰化,从而调节大肠杆菌的代谢。

Zhang, Jianji; Zang, Yong; Yu, Zhiqing; Zhao, Fei; Wu, Jingya; Wang, Yuhan; Wang, Aiyuan; Zhai, Guijin; Han, Yanpu; Chen, Chen; Ye, Jinjun; Dong, Hanyang; Zhang, Kai

SETD2 suppresses tumorigenesis in a KRAS(G12C)-driven lung cancer model, and its catalytic activity is regulated by histone acetylation.

SETD2 在 KRAS(G12C) 驱动的肺癌模型中抑制肿瘤发生,其催化活性受组蛋白乙酰化的调控

Mack Ricardo J, Flores Natasha M, Fox Geoffrey C, Dong Hanyang, Cebeci Metehan, Hausmann Simone, Chasan Tourkian, Dowen Jill M, Strahl Brian D, Mazur Pawel K, Gozani Or

SETD2 suppresses tumorigenesis in a KRAS(G12C)-driven lung cancer model and its catalytic activity is regulated by histone acetylation.

SETD2 在 KRAS(G12C) 驱动的肺癌模型中抑制肿瘤发生,其催化活性受组蛋白乙酰化的调控

Mack Ricardo J, Flores Natasha M, Fox Geoffrey C, Dong Hanyang, Cebeci Metehan, Hausmann Simone, Chasan Tourkian, Dowen Jill M, Strahl Brian D, Mazur Pawel K, Gozani Or