日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Kikuchi-Fujimoto disease: the quandary continues

菊池-藤本病:难题仍在继续

Caesar-Peterson, Shannon; Yoon, Dosuk; Tulla, Katrina; Nahidi, Seyed M; Tickoo, Sharang; Sheikh, Mudnia; Depaz, Hector

MG53 Preserves Neuromuscular Junction Integrity and Alleviates ALS Disease Progression

MG53 保持神经肌肉接头完整性并缓解 ALS 疾病进展

Jianxun Yi, Ang Li, Xuejun Li, Kiho Park, Xinyu Zhou, Frank Yi, Yajuan Xiao, Dosuk Yoon, Tao Tan, Lyle W Ostrow, Jianjie Ma, Jingsong Zhou

Superior gluteal artery pseudoaneurysm after a gunshot wound to the buttock: A case

臀部枪伤后臀上动脉假性动脉瘤:一例报告

Coste, Marine; Yoon, Dosuk; Noory, Mary; Roudnitsky, Valery

Microglial activation in an amyotrophic lateral sclerosis-like model caused by Ranbp2 loss and nucleocytoplasmic transport impairment in retinal ganglion neurons

由 Ranbp2 缺失和视网膜神经节神经元核质运输障碍引起的肌萎缩侧索硬化症样模型中的小胶质细胞活化

Kyoung-In Cho, Dosuk Yoon, Minzhong Yu, Neal S Peachey, Paulo A Ferreira

Impairments in age-dependent ubiquitin proteostasis and structural integrity of selective neurons by uncoupling Ran GTPase from the Ran-binding domain 3 of Ranbp2 and identification of novel mitochondrial isoforms of ubiquitin-conjugating enzyme E2I (ubc9) and Ranbp2

通过将 Ran GTPase 与 Ranbp2 的 Ran 结合域 3 分离,并鉴定出泛素结合酶 E2I (ubc9) 和 Ranbp2 的新型线粒体异构体,可导致年龄依赖性泛素蛋白稳态和选择性神经元结构完整性受损

Hemangi Patil, Dosuk Yoon, Reshma Bhowmick, Yunfei Cai, Kyoung-In Cho, Paulo A Ferreira

ROS-related mitochondrial dysfunction in skeletal muscle of an ALS mouse model during the disease progression

ALS 小鼠模型在疾病进展过程中骨骼肌中 ROS 相关的线粒体功能障碍

Yajuan Xiao, Chehade Karam, Jianxun Yi, Lin Zhang, Xuejun Li, Dosuk Yoon, Huan Wang, Kamal Dhakal, Paul Ramlow, Tian Yu, Zhaohui Mo, Jianjie Ma, Jingsong Zhou

Loss of Ranbp2 in motoneurons causes disruption of nucleocytoplasmic and chemokine signaling, proteostasis of hnRNPH3 and Mmp28, and development of amyotrophic lateral sclerosis-like syndromes

运动神经元中 Ranbp2 的缺失会导致核质和趋化因子信号传导中断、hnRNPH3 和 Mmp28 的蛋白质稳态以及肌萎缩侧索硬化症样综合征的发展

Kyoung-In Cho, Dosuk Yoon, Sunny Qiu, Zachary Danziger, Warren M Grill, William C Wetsel, Paulo A Ferreira

Uncoupling phototoxicity-elicited neural dysmorphology and death by insidious function and selective impairment of Ran-binding protein 2 (Ranbp2)

通过 Ran 结合蛋白 2 (Ranbp2) 的隐匿功能和选择性损伤,解耦光毒性引起的神经形态异常和死亡

Cho, Kyoung-in; Haney, Victoria; Yoon, Dosuk; Hao, Yin; Ferreira, Paulo A

Selective impairment of a subset of Ran-GTP-binding domains of ran-binding protein 2 (Ranbp2) suffices to recapitulate the degeneration of the retinal pigment epithelium (RPE) triggered by Ranbp2 ablation

选择性损伤 Ran 结合蛋白 2 (Ranbp2) 的 Ran-GTP 结合域子集,足以重现由 Ranbp2 消融引发的视网膜色素上皮 (RPE) 退化

Hemangi Patil, Arjun Saha, Eugene Senda, Kyoung-in Cho, MdEmdadul Haque, Minzhong Yu, Sunny Qiu, Dosuk Yoon, Ying Hao, Neal S Peachey, Paulo A Ferreira

Differential loss of prolyl isomerase or chaperone activity of Ran-binding protein 2 (Ranbp2) unveils distinct physiological roles of its cyclophilin domain in proteostasis

Ran结合蛋白2(Ranbp2)的脯氨酰异构酶或分子伴侣活性的差异性丧失揭示了其环丝氨酸结构域在蛋白质稳态中的不同生理作用

Kyoung-in Cho, Hemangi Patil, Eugene Senda, Jessica Wang, Haiqing Yi, Sunny Qiu, Dosuk Yoon, Minzhong Yu, Andrew Orry, Neal S Peachey, Paulo A Ferreira