Doyle Timothy M相关文献与解析，生知库 | 链接生命科学的每一步

Schafer, Rachel M; Giancotti, Luigino A; Chrivia, John C; Li, Ying; Mufti, Fatma; Kufer, Thomas A; Zhang, Jinsong; Doyle, Timothy M; Salvemini, Daniela

NOD2

发表日期：2025

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Mitochondrial A(3) Adenosine Receptor as a Mechanism for the Protective Effects of A(3)AR Agonists on Chemotherapy-Induced Neuropathic Pain.

线粒体 A(3) 腺苷受体作为 A(3)AR 激动剂对化疗引起的神经性疼痛的保护作用机制

期刊:Journal of Neuroscience

影响因子:4

doi:10.1523/JNEUROSCI.1268-24.2024

Doyle Timothy M, Janes Kali, Xiao Wen Hua, Kolar Grant R, Luecke Hans F, Gratton Michael Anne, Tosh Dilip K, Jacobson Kenneth A, Bennett Gary J, Salvemini Daniela

Targeting neuroinflammation in neuropathic pain and opioid use

针对神经性疼痛和阿片类药物使用中的神经炎症进行治疗

期刊:Journal of Experimental Medicine

影响因子:10.6

doi:10.1084/jem.20221244

Salvemini, Daniela; Doyle, Timothy M

Sphingosine-1-phosphate receptor 1 activation in the central nervous system drives cisplatin-induced cognitive impairment

中枢神经系统中鞘氨醇-1-磷酸受体1的激活驱动顺铂引起的认知障碍

期刊:Journal of Clinical Investigation

影响因子:13.6

doi:10.1172/JCI157738

Squillace, Silvia; Niehoff, Michael L; Doyle, Timothy M; Green, Michael; Esposito, Emanuela; Cuzzocrea, Salvatore; Arnatt, Christopher K; Spiegel, Sarah; Farr, Susan A; Salvemini, Daniela

Mini-Review: Mitochondrial dysfunction and chemotherapy-induced neuropathic pain

简评：线粒体功能障碍与化疗引起的神经性疼痛

期刊:Neuroscience Letters

影响因子:2

doi:10.1016/j.neulet.2021.136087

Doyle, Timothy M; Salvemini, Daniela

线粒体

发表日期：2021

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GPR160 de-orphanization reveals critical roles in neuropathic pain in rodents

GPR160去孤儿化揭示其在啮齿动物神经性疼痛中的关键作用

期刊:Journal of Clinical Investigation

影响因子:13.6

doi:10.1172/JCI133270

Yosten, Gina Lc; Harada, Caron M; Haddock, Chris; Giancotti, Luigino Antonio; Kolar, Grant R; Patel, Ryan; Guo, Chun; Chen, Zhoumou; Zhang, Jinsong; Doyle, Timothy M; Dickenson, Anthony H; Samson, Willis K; Salvemini, Daniela

发表日期：2020

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Chronic Morphine-Induced Changes in Signaling at the A(3) Adenosine Receptor Contribute to Morphine-Induced Hyperalgesia, Tolerance, and Withdrawal

慢性吗啡引起的A(3)腺苷受体信号传导变化是导致吗啡诱发痛觉过敏、耐受性和戒断反应的原因之一。

期刊:Journal of Pharmacology and Experimental Therapeutics

影响因子:3.8

doi:10.1124/jpet.120.000004

Doyle, Timothy M; Largent-Milnes, Tally M; Chen, Zhoumou; Staikopoulos, Vasiliki; Esposito, Emanuela; Dalgarno, Rebecca; Fan, Churmy; Tosh, Dilip K; Cuzzocrea, Salvatore; Jacobson, Kenneth A; Trang, Tuan; Hutchinson, Mark R; Bennett, Gary J; Vanderah, Todd W; Salvemini, Daniela

信号转导

发表日期：2020

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Acute hypoxia decreases E. coli LPS-induced cytokine production and NF-kappaB activation in alveolar macrophages

急性缺氧会降低大肠杆菌LPS诱导的肺泡巨噬细胞细胞因子产生和NF-κB活化。

期刊:Respiratory Physiology & Neurobiology

影响因子:1.6

doi:10.1016/j.resp.2010.05.006

Matuschak, George M; Nayak, Ravi; Doyle, Timothy M; Lechner, Andrew J

CARTp/GPR160 mediates behavioral hypersensitivities in mice through NOD2

CARTp/GPR160 通过 NOD2 介导小鼠的行为过敏

Mitochondrial A(3) Adenosine Receptor as a Mechanism for the Protective Effects of A(3)AR Agonists on Chemotherapy-Induced Neuropathic Pain.

线粒体 A(3) 腺苷受体作为 A(3)AR 激动剂对化疗引起的神经性疼痛的保护作用机制

Targeting neuroinflammation in neuropathic pain and opioid use

针对神经性疼痛和阿片类药物使用中的神经炎症进行治疗

Sphingosine-1-phosphate receptor 1 activation in the central nervous system drives cisplatin-induced cognitive impairment

中枢神经系统中鞘氨醇-1-磷酸受体1的激活驱动顺铂引起的认知障碍

Mini-Review: Mitochondrial dysfunction and chemotherapy-induced neuropathic pain

简评：线粒体功能障碍与化疗引起的神经性疼痛

GPR160 de-orphanization reveals critical roles in neuropathic pain in rodents

GPR160去孤儿化揭示其在啮齿动物神经性疼痛中的关键作用

Chronic Morphine-Induced Changes in Signaling at the A(3) Adenosine Receptor Contribute to Morphine-Induced Hyperalgesia, Tolerance, and Withdrawal

慢性吗啡引起的A(3)腺苷受体信号传导变化是导致吗啡诱发痛觉过敏、耐受性和戒断反应的原因之一。

Acute hypoxia decreases E. coli LPS-induced cytokine production and NF-kappaB activation in alveolar macrophages

急性缺氧会降低大肠杆菌LPS诱导的肺泡巨噬细胞细胞因子产生和NF-κB活化。