日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Optogenetic restoration of neuron subtype-specific cortical activity ameliorates motor deficits in Huntington's Disease mice

光遗传学方法恢复神经元亚型特异性皮层活动可改善亨廷顿病小鼠的运动障碍。

Blumenstock, Sonja; Arakelyan, David; Del Grosso, Nicholas; Schneider, Sonja; Shao, Yufeng; Gjoni, Enida; Klein, Rüdiger; Dudanova, Irina; Komiyama, Takaki

The AAA+ chaperone VCP disaggregates Tau fibrils and generates aggregate seeds in a cellular system

AAA+ 分子伴侣 VCP 分解 Tau 纤维并在细胞系统中产生聚集种子

Itika Saha, Patricia Yuste-Checa, Miguel Da Silva Padilha, Qiang Guo, Roman Körner, Hauke Holthusen, Victoria A Trinkaus, Irina Dudanova, Rubén Fernández-Busnadiego, Wolfgang Baumeister, David W Sanders, Saurabh Gautam, Marc I Diamond, F Ulrich Hartl, Mark S Hipp

Neuroprotective effects of hepatoma-derived growth factor in models of Huntington's disease

肝癌衍生生长因子在亨廷顿病模型中的神经保护作用

Kerstin Voelkl,Sara Gutiérrez-Ángel,Sophie Keeling,Seda Koyuncu,Miguel da Silva Padilha,Dennis Feigenbutz,Thomas Arzberger,David Vilchez,Rüdiger Klein,Irina Dudanova

Biosensors for Studying Neuronal Proteostasis

用于研究神经元蛋白质稳态的生物传感器

Dudanova, Irina

Distinct histological alterations of cortical interneuron types in mouse models of Huntington's disease

亨廷顿氏病小鼠模型中皮质中间神经元类型的不同组织学改变

Kerstin Voelkl, Elena Katharina Schulz-Trieglaff, Rüdiger Klein, Irina Dudanova

The extracellular chaperone Clusterin enhances Tau aggregate seeding in a cellular model

细胞外分子伴侣 Clusterin 在细胞模型中增强 Tau 聚集体的播种

Patricia Yuste-Checa, Victoria A Trinkaus, Irene Riera-Tur, Rahmi Imamoglu, Theresa F Schaller, Huping Wang, Irina Dudanova, Mark S Hipp, Andreas Bracher, F Ulrich Hartl

In situ architecture of neuronal α-Synuclein inclusions

神经元 α-突触核蛋白内含物的原位结构

Victoria A Trinkaus, Irene Riera-Tur, Antonio Martínez-Sánchez, Felix J B Bäuerlein, Qiang Guo, Thomas Arzberger, Wolfgang Baumeister, Irina Dudanova, Mark S Hipp, F Ulrich Hartl, Rubén Fernández-Busnadiego

Fluc-EGFP reporter mice reveal differential alterations of neuronal proteostasis in aging and disease

Fluc-EGFP 报告小鼠揭示衰老和疾病过程中神经元蛋白稳态的差异改变

Sonja Blumenstock, Elena Katharina Schulz-Trieglaff, Kerstin Voelkl, Anna-Lena Bolender, Paul Lapios, Jana Lindner, Mark S Hipp, F Ulrich Hartl, Rüdiger Klein, Irina Dudanova

Amyloid-like aggregating proteins cause lysosomal defects in neurons via gain-of-function toxicity

淀粉样聚集蛋白通过功能获得性毒性导致神经元溶酶体缺陷

Irene Riera-Tur,Tillman Schäfer,Daniel Hornburg,Archana Mishra,Miguel da Silva Padilha,Lorena Fernández-Mosquera,Dennis Feigenbutz,Patrick Auer,Matthias Mann,Wolfgang Baumeister,Rüdiger Klein,Felix Meissner,Nuno Raimundo,Rubén Fernández-Busnadiego,Irina Dudanova

Cortical and Striatal Circuits in Huntington's Disease

亨廷顿病中的皮层和纹状体回路

Blumenstock, Sonja; Dudanova, Irina