Dues Dylan J相关文献与解析，生知库 | 链接生命科学的每一步

Hippocampal subfield vulnerability to α-synuclein pathology precedes neurodegeneration and cognitive dysfunction

海马亚区对α-突触核蛋白病理的易感性先于神经退行性变和认知功能障碍。

期刊:Npj Parkinsons Disease

影响因子:8.2

doi:10.1038/s41531-023-00574-1

Dues, Dylan J; Nguyen, An Phu Tran; Becker, Katelyn; Ma, Jiyan; Moore, Darren J

LRRK2 and Protein Aggregation in Parkinson's Disease: Insights From Animal Models

LRRK2与帕金森病中的蛋白质聚集：来自动物模型的启示

期刊:Frontiers in Neuroscience

影响因子:3.2

doi:10.3389/fnins.2020.00719

Dues, Dylan J; Moore, Darren J

Mitochondrial unfolded protein response transcription factor ATFS-1 promotes longevity in a long-lived mitochondrial mutant through activation of stress response pathways

线粒体未折叠蛋白反应转录因子ATFS-1通过激活应激反应通路促进长寿线粒体突变体的寿命

期刊:BMC Biology

影响因子:4.5

doi:10.1186/s12915-018-0615-3

Wu, Ziyun; Senchuk, Megan M; Dues, Dylan J; Johnson, Benjamin K; Cooper, Jason F; Lew, Leira; Machiela, Emily; Schaar, Claire E; DeJonge, Heather; Blackwell, T Keith; Van Raamsdonk, Jeremy M

Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans

活性氧激活DAF-16/FOXO有助于秀丽隐杆线虫长寿线粒体突变体的寿命延长。

期刊:PLoS Genetics

影响因子:3.7

doi:10.1371/journal.pgen.1007268

Senchuk, Megan M; Dues, Dylan J; Schaar, Claire E; Johnson, Benjamin K; Madaj, Zachary B; Bowman, Megan J; Winn, Mary E; Van Raamsdonk, Jeremy M

Uncoupling of oxidative stress resistance and lifespan in long-lived isp-1 mitochondrial mutants in Caenorhabditis elegans.

秀丽隐杆线虫长寿 isp-1 线粒体突变体中氧化应激抵抗力与寿命的脱钩

期刊:Free Radical Biology and Medicine

影响因子:8.2

doi:10.1016/j.freeradbiomed.2017.04.004

Dues Dylan J, Schaar Claire E, Johnson Benjamin K, Bowman Megan J, Winn Mary E, Senchuk Megan M, Van Raamsdonk Jeremy M

Delaying aging is neuroprotective in Parkinson's disease: a genetic analysis in C. elegans models

延缓衰老对帕金森病具有神经保护作用：秀丽隐杆线虫模型遗传分析

期刊:Npj Parkinsons Disease

影响因子:8.2

doi:10.1038/npjparkd.2015.22

Cooper, Jason F; Dues, Dylan J; Spielbauer, Katie K; Machiela, Emily; Senchuk, Megan M; Van Raamsdonk, Jeremy M

Pathological Î±-synuclein elicits granulovacuolar degeneration independent of tau.

病理性α-突触核蛋白可引起与tau蛋白无关的颗粒空泡变性

Hippocampal subfield vulnerability to α-synuclein pathology precedes neurodegeneration and cognitive dysfunction

海马亚区对α-突触核蛋白病理的易感性先于神经退行性变和认知功能障碍。

LRRK2 and Protein Aggregation in Parkinson's Disease: Insights From Animal Models

LRRK2与帕金森病中的蛋白质聚集：来自动物模型的启示

Mitochondrial unfolded protein response transcription factor ATFS-1 promotes longevity in a long-lived mitochondrial mutant through activation of stress response pathways

线粒体未折叠蛋白反应转录因子ATFS-1通过激活应激反应通路促进长寿线粒体突变体的寿命

Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans

活性氧激活DAF-16/FOXO有助于秀丽隐杆线虫长寿线粒体突变体的寿命延长。

Uncoupling of oxidative stress resistance and lifespan in long-lived isp-1 mitochondrial mutants in Caenorhabditis elegans.

秀丽隐杆线虫长寿 isp-1 线粒体突变体中氧化应激抵抗力与寿命的脱钩

Delaying aging is neuroprotective in Parkinson's disease: a genetic analysis in C. elegans models

延缓衰老对帕金森病具有神经保护作用：秀丽隐杆线虫模型遗传分析