日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Somatic inactivating PTPRJ mutations and dysregulated pathways identified in canine malignant melanoma by integrated comparative genomic analysis.

通过综合比较基因组分析,在犬恶性黑色素瘤中发现了体细胞失活的 PTPRJ 突变和失调通路。

Hendricks William P D, Zismann Victoria, Sivaprakasam Karthigayini, Legendre Christophe, Poorman Kelsey, Tembe Waibhav, Perdigones Nieves, Kiefer Jeffrey, Liang Winnie, DeLuca Valerie, Stark Mitchell, Ruhe Alison, Froman Roe, Duesbery Nicholas S, Washington Megan, Aldrich Jessica, Neff Mark W, Huentelman Matthew J, Hayward Nicholas, Brown Kevin, Thamm Douglas, Post Gerald, Khanna Chand, Davis Barbara, Breen Matthew, Sekulic Alexander, Trent Jeffrey M

MEK genomics in development and disease

MEK基因组学在发育和疾病中的作用

Bromberg-White, Jennifer L; Andersen, Nicholas J; Duesbery, Nicholas S

MEK2 is sufficient but not necessary for proliferation and anchorage-independent growth of SK-MEL-28 melanoma cells.

MEK2 对于 SK-MEL-28 黑色素瘤细胞的增殖和非锚定依赖性生长是充分的,但并非必需的

Lee Chih-Shia, Dykema Karl J, Hawkins Danielle M, Cherba David M, Webb Craig P, Furge Kyle A, Duesbery Nicholas S

Inhibition of tumor angiogenesis by the matrix metalloproteinase-activated anthrax lethal toxin in an orthotopic model of anaplastic thyroid carcinoma

在原位未分化型甲状腺癌模型中,基质金属蛋白酶激活的炭疽致死毒素抑制肿瘤血管生成

Alfano, Randall W; Leppla, Stephen H; Liu, Shihui; Bugge, Thomas H; Ortiz, Janelle M; Lairmore, Terry C; Duesbery, Nicholas S; Mitchell, Ian C; Nwariaku, Fiemu; Frankel, Arthur E

LRP5 and LRP6 are not required for protective antigen-mediated internalization or lethality of anthrax lethal toxin.

LRP5 和 LRP6 不是炭疽致死毒素通过抗原介导的保护性内化或致死作用所必需的

Young John J, Bromberg-White Jennifer L, Zylstra Cassandra, Church Joseph T, Boguslawski Elissa, Resau James H, Williams Bart O, Duesbery Nicholas S