Dustin C Hancks相关文献与解析，生知库 | 链接生命科学的每一步

Emily A Rex ，Dahee Seo ，Sruthi Chappidi ，Chelsea Pinkham ，Sabrynna Brito Oliveira ，Aaron Embry ，David Heisler ，Yang Liu ，Moiz Munir ，Karolin Luger ，Neal M Alto ，Flávio Guimarães da Fonseca ，Robert Orchard ，Dustin C Hancks ，Don B Gammon

信号转导

微生物学

发表日期：2024

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Metabolic reprogramming tips vaccinia virus infection outcomes by stabilizing interferon-γ induced IRF1

代谢重编程通过稳定干扰素-γ 诱导的 IRF1 来改善痘苗病毒感染结果

期刊:PLoS Pathogens

影响因子:5.5

doi:10.1371/journal.ppat.1012673

Tyron Chang, Jessica Alvarez, Sruthi Chappidi, Stacey Crockett, Mahsa Sorouri, Robert C Orchard, Dustin C Hancks

Norovirus MLKL-like protein initiates cell death to induce viral egress

诺如病毒 MLKL 样蛋白启动细胞死亡，诱导病毒外泄

期刊:Nature

影响因子:50.5

doi:10.1038/s41586-023-05851-w

Guoxun Wang, Di Zhang, Robert C Orchard, Dustin C Hancks, Tiffany A Reese

Species-specific self-DNA detection mechanisms by mammalian cyclic GMP-AMP synthases

哺乳动物环状GMP-AMP合成酶的物种特异性自身DNA检测机制

期刊:Science Immunology

影响因子:17.6

doi:10.1126/sciimmunol.abp9765.

Kenta Mosallanejad,Stephanie N Kennedy,Kristin M Bahleda,Kailey M Slavik,Wen Zhou,Apurva A Govande,Dustin C Hancks,Philip J Kranzusch,Jonathan C Kagan

发表日期：2023

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Evolutionary Profile for (Host and Viral) MLKL Indicates Its Activities as a Battlefront for Extensive Counteradaptation

MLKL（宿主和病毒）的进化概况表明其活动是广泛反适应的战场

期刊:Molecular Biology and Evolution

影响因子:11

doi:10.1093/molbev/msab256

Suzette N Palmer, Sruthi Chappidi, Chelsea Pinkham, Dustin C Hancks

微生物学

发表日期：2021

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Signatures of host-pathogen evolutionary conflict reveal MISTR-A conserved MItochondrial STress Response network

宿主-病原体进化冲突的特征揭示了MISTR-A保守的线粒体应激反应网络

期刊:PLoS Biology

影响因子:7.8

doi:10.1371/journal.pbio.3001045

Mahsa Sorouri,Tyron Chang,Palmy Jesudhasan,Chelsea Pinkham,Nels C Elde,Dustin C Hancks

cGAS-mediated stabilization of IFI16 promotes innate signaling during herpes simplex virus infection

cGAS 介导的 IFI16 稳定化促进单纯疱疹病毒感染期间的先天信号传导

期刊:Proceedings of the National Academy of Sciences of the United States of America

影响因子:

doi:10.1073/pnas.1424637112

Megan H Orzalli, Nicole M Broekema, Benjamin A Diner, Dustin C Hancks, Nels C Elde, Ileana M Cristea, David M Knipe

FEAR antiviral response pathway is independent of interferons and countered by poxvirus proteins

FEAR抗病毒反应通路不依赖于干扰素，并会被痘病毒蛋白拮抗。

Metabolic reprogramming tips vaccinia virus infection outcomes by stabilizing interferon-γ induced IRF1

代谢重编程通过稳定干扰素-γ 诱导的 IRF1 来改善痘苗病毒感染结果

Norovirus MLKL-like protein initiates cell death to induce viral egress

诺如病毒 MLKL 样蛋白启动细胞死亡，诱导病毒外泄

Species-specific self-DNA detection mechanisms by mammalian cyclic GMP-AMP synthases

哺乳动物环状GMP-AMP合成酶的物种特异性自身DNA检测机制

Evolutionary Profile for (Host and Viral) MLKL Indicates Its Activities as a Battlefront for Extensive Counteradaptation

MLKL（宿主和病毒）的进化概况表明其活动是广泛反适应的战场

Signatures of host-pathogen evolutionary conflict reveal MISTR-A conserved MItochondrial STress Response network

宿主-病原体进化冲突的特征揭示了MISTR-A保守的线粒体应激反应网络

cGAS-mediated stabilization of IFI16 promotes innate signaling during herpes simplex virus infection

cGAS 介导的 IFI16 稳定化促进单纯疱疹病毒感染期间的先天信号传导