Dutca Laura M相关文献与解析，生知库 | 链接生命科学的每一步

Occupational-like organophosphate exposure disrupts microglia and accelerates deficits in a rat model of Alzheimer's disease

职业性有机磷酸酯暴露会破坏小胶质细胞，并加速阿尔茨海默病大鼠模型的病情恶化。

期刊:npj Aging and Mechanisms of Disease

影响因子:5.4

doi:10.1038/s41514-018-0033-3

Voorhees, Jaymie R; Remy, Matthew T; Erickson, Claire M; Dutca, Laura M; Brat, Daniel J; Pieper, Andrew A

(-)-P7C3-S243 Protects a Rat Model of Alzheimer's Disease From Neuropsychiatric Deficits and Neurodegeneration Without Altering Amyloid Deposition or Reactive Glia

(-)-P7C3-S243 可保护阿尔茨海默病大鼠模型免受神经精神缺陷和神经退行性变的影响，且不改变淀粉样蛋白沉积或反应性胶质细胞。

期刊:Biological Psychiatry

影响因子:9

doi:10.1016/j.biopsych.2017.10.023

Voorhees, Jaymie R; Remy, Matthew T; Cintrón-Pérez, Coral J; El Rassi, Eli; Khan, Michael Z; Dutca, Laura M; Yin, Terry C; McDaniel, Latisha N; Williams, Noelle S; Brat, Daniel J; Pieper, Andrew A

RetFM-J, an ImageJ-based module for automated counting and quantifying features of nuclei in retinal whole-mounts

RetFM-J，一个基于 ImageJ 的模块，用于自动计数和量化视网膜全装片中细胞核的特征。

期刊:Experimental Eye Research

影响因子:2.7

doi:10.1016/j.exer.2015.07.020

Hedberg-Buenz, Adam; Christopher, Mark A; Lewis, Carly J; Meyer, Kacie J; Rudd, Danielle S; Dutca, Laura M; Wang, Kai; Garvin, Mona K; Scheetz, Todd E; Abràmoff, Michael D; Harper, Matthew M; Anderson, Michael G

细胞生物学

发表日期：2016

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When ribosomes go bad: diseases of ribosome biogenesis

核糖体功能异常：核糖体生物合成疾病

期刊:Molecular Biosystems

影响因子:

doi:10.1039/b919670f

Freed, Emily F; Bleichert, Franziska; Dutca, Laura M; Baserga, Susan J

发表日期：2010

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Disruption of circadian intraocular pressure fluctuations in mice by the Lyst beige-J mutation

Lyst beige-J突变体对小鼠昼夜节律性眼内压波动的干扰

Yeast pre-rRNA is processed at the A' site

酵母前体rRNA在A'位点进行加工。

Axonopathy precedes cell death in ocular damage mediated by blast exposure.

爆炸冲击波引起的眼部损伤中，轴突病变先于细胞死亡发生

Occupational-like organophosphate exposure disrupts microglia and accelerates deficits in a rat model of Alzheimer's disease

职业性有机磷酸酯暴露会破坏小胶质细胞，并加速阿尔茨海默病大鼠模型的病情恶化。

(-)-P7C3-S243 Protects a Rat Model of Alzheimer's Disease From Neuropsychiatric Deficits and Neurodegeneration Without Altering Amyloid Deposition or Reactive Glia

(-)-P7C3-S243 可保护阿尔茨海默病大鼠模型免受神经精神缺陷和神经退行性变的影响，且不改变淀粉样蛋白沉积或反应性胶质细胞。

RetFM-J, an ImageJ-based module for automated counting and quantifying features of nuclei in retinal whole-mounts

RetFM-J，一个基于 ImageJ 的模块，用于自动计数和量化视网膜全装片中细胞核的特征。

When ribosomes go bad: diseases of ribosome biogenesis

核糖体功能异常：核糖体生物合成疾病