日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Selective vulnerability of the aging cholinergic system to amyloid pathology revealed by induced APP overexpression.

诱导 APP 过表达揭示了衰老胆碱能系统对淀粉样蛋白病理的选择性脆弱性。

Xie Kan, Ryan Devon, Schröder Susanne, Freund Lena, Bonn Stefan, Zhou Yu, Ehninger Dan

Randomized Comparison of Cardiotoxicity With 60 Versus 90 mg Daunorubicin in AML Induction Therapy

一项随机对照研究比较了急性髓系白血病诱导治疗中 60 mg 与 90 mg 柔红霉素的心脏毒性

Dendorfer, Stefan Markus; Schmidt-Brücken, Katharina; Kramer, Michael; Steffen, Björn; Schliemann, Christoph; Mikesch, Jan-Henrik; Alakel, Nael; Herbst, Regina; Hänel, Mathias; Hanoun, Maher; Kaufmann, Martin; Weinbergerova, Barbora; Schäfer-Eckart, Kerstin; Sauer, Tim; Neubauer, Andreas; Burchert, Andreas; Baldus, Claudia D; Mertová, Jolana; Jost, Edgar; Niemann, Dirk; Novák, Jan; Krause, Stefan W; Scholl, Sebastian; Hochhaus, Andreas; Held, Gerhard; Szotkowski, Tomáš; Rank, Andreas; Schmid, Christoph; Fransecky, Lars; Kayser, Sabine; Schaich, Markus; Fiebig, Frank; Haake, Annett; Schetelig, Johannes; Middeke, Jan Moritz; Stölzel, Friedrich; Platzbecker, Uwe; Thiede, Christian; Müller-Tidow, Carsten; Berdel, Wolfgang E; Ehninger, Gerhard; Mayer, Jiri; Serve, Hubert; Bornhäuser, Martin; Röllig, Christoph

Unraveling the intersection of aging and Parkinson's disease: a collaborative roadmap for advancing research models

揭示衰老与帕金森病之间的交集:推进研究模型的合作路线图

Schmidt, M Y; Cuervo, A M; Double, K L; Ehninger, D; Goldberg, M S; Harvey, K; Hoeijmakers, J H J; Luk, K C; Mastroberardino, P G; Moore, D J; Niedernhofer, L J; Trudeau, L É; Jurk, D; Andersen, J K; Bellantuono, I

Deciphering the Transcriptomic Signatures of Aging Across Organs in Mice

解读小鼠各器官衰老转录组特征

Morsy, Sarah; Scifo, Enzo; Xie, Kan; Schaaf, Kristina; Russ, Jenny; Paulusch, Stefan; De Domenico, Elena; Salomoni, Paolo; Bano, Daniele; Ehninger, Dan

BCL-11 enables adaptive stress responses to environmental challenges

BCL-11能够使人体对环境挑战产生适应性应激反应

Niedworok, Patrizia; Ciliberti, Rossella Erminia; Xie, Beijia; Mathew, Amal John; Jussila, Benjamin; Lawson, Jennifer A; De Domenico, Elena; Paulusch, Stefan; Beyer, Marc; Nicotera, Pierluigi; Aziz, N Ahmad; Ehninger, Dan; Blaess, Sandra; Bano, Daniele

PRDM16 expression is an independent prognostic factor in AML with the double-mutant NPM1/FLT3-ITD genotype

PRDM16表达是NPM1/FLT3-ITD双突变基因型AML的独立预后因素

Stasik, Sebastian; Eckardt, Jan-Niklas; Röllig, Christoph; Baldus, Claudia D; Serve, Hubert; Müller-Tidow, Carsten; Schäfer-Eckart, Kerstin; Kaufmann, Martin; Krause, Stefan W; Hänel, Mathias; Neubauer, Andreas; Ehninger, Gerhard; Platzbecker, Uwe; Bornhäuser, Martin; Schetelig, Johannes; Middeke, Jan M; Thiede, Christian

Disease risk but not remission status determines transplant outcomes in AML: long-term outcomes of the ASAP trial

急性髓系白血病(AML)的移植结局取决于疾病风险而非缓解状态:ASAP试验的长期结果

Stelljes, Matthias; Middeke, Jan Moritz; Bug, Gesine; Wagner-Drouet, Eva-Maria; Müller, Lutz P; Schmid, Christoph; Krause, Stefan W; Bethge, Wolfgang; Jost, Edgar; Platzbecker, Uwe; Klein, Stefan A; Niederland, Judith; Kaufmann, Martin; Schäfer-Eckart, Kerstin; Baldauf, Henning; Stölzel, Friedrich; Trost, Sarah; Röllig, Christoph; von Bonin, Malte; Egger-Heidrich, Katharina; Kunadt, Desiree; Steffen, Björn; Hauptrock, Beate; Schliemann, Christoph; Sockel, Katja; Lang, Fabian; Kriege, Oliver; Schaffrath, Judith; Reicherts, Christian; Berdel, Wolfgang E; Serve, Hubert; Ehninger, Gerhard; Schmidt, Alexander H; Mikesch, Jan-Henrik; Bornhäuser, Martin; Schetelig, Johannes

Interaction with AK2A links AIFM1 to cellular energy metabolism.

AIFM1 与 AK2A 相互作用,从而将 AIFM1 与细胞能量代谢联系起来

Rothemann Robin Alexander, Pavlenko Egor, Mondal Mrityunjoy, Gerlich Sarah, Grobushkin Pavel, Mostert Sebastian, Racho Julia, Weiss Konstantin, Stobbe Dylan, Stillger Katharina, Lapacz Kim, Salscheider Silja Lucia, Petrungaro Carmelina, Ehninger Dan, Nguyen Thi Hoang Duong, Dengjel Jörn, Neundorf Ines, Bano Daniele, Poepsel Simon, Riemer Jan

TMEM65-dependent Ca2+ extrusion safeguards mitochondrial homeostasis

TMEM65依赖的Ca2+外排维持线粒体稳态

Massimo Vetralla,Lena Wischhof #,Asrat Kahsay #,Vanessa Cadenelli,Enzo Scifo,Beijia Xie,Miriana Sbrissa,Maëlle Sandhira Habert,Dan Ehninger,Rosario Rizzuto,Daniele Bano,Diego De Stefani

Loss of Ten1 in mice induces telomere shortening and models human dyskeratosis congenita.

小鼠中 Ten1 的缺失会导致端粒缩短,并模拟人类先天性角化不良症

Sanz-Moreno Adrián, Becker Lore, Xie Kan, da Silva-Buttkus Patricia, Dragano Nathalia R V, Aguilar-Pimentel Antonio, Amarie Oana V, Calzada-Wack Julia, Kraiger Markus, Leuchtenberger Stefanie, Seisenberger Claudia, Marschall Susan, Rathkolb Birgit, Scifo Enzo, Liu Ting, Thanabalasingam Anoja, Sanchez-Vazquez Raul, Martinez Paula, Blasco Maria A, Savage Sharon A, Fuchs Helmut, Ehninger Dan, Gailus-Durner Valérie, de Angelis Martin Hrabê