Eike-Benjamin相关文献与解析，生知库 | 链接生命科学的每一步

Eike-Benjamin Braune #, Felix Geist #, Xiaojia Tang, Krishna Kalari, Judy Boughey, Liewei Wang, Roberto A Leon-Ferre, Antonino B D'Assoro, James N Ingle, Matthew P Goetz, Julian Kreis, Kang Wang, Theodoros Foukakis, Anita Seshire, Dirk Wienke, Urban Lendahl

信号转导

肿瘤

乳腺癌

Left ventricular hypertrophy and metabolic resetting in the Notch3-deficient adult mouse heart

Notch3 缺陷成年小鼠心脏左心室肥大和代谢重置

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-023-42010-7

Francesca Del Gaudio #, Dongli Liu #, Maarja Andaloussi Mäe, Eike-Benjamin Braune, Emil M Hansson, Qing-Dong Wang, Christer Betsholtz, Urban Lendahl

Roles of Notch Signaling in the Tumor Microenvironment

Notch信号通路在肿瘤微环境中的作用

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms23116241

D'Assoro, Antonino B; Leon-Ferre, Roberto; Braune, Eike-Benjamin; Lendahl, Urban

Novel Cysteine-Sparing Hypomorphic NOTCH3 A1604T Mutation Observed in a Family With Migraine and White Matter Lesions

在患有偏头痛和白质病变的家族中发现了一种新型的保留半胱氨酸的低活性NOTCH3 A1604T突变

期刊:Neurology-Genetics

影响因子:3.7

doi:10.1212/NXG.0000000000000584

Arnardottir, Snjolaug; Del Gaudio, Francesca; Klironomos, Stefanos; Braune, Eike-Benjamin; Lombraña, Ariane Araujo; Oliveira, Daniel V; Jin, Shaobo; Karlström, Helena; Lendahl, Urban; Sjöstrand, Christina

Notch signalling regulates epibranchial placode patterning and segregation

Notch 信号调节鳃上斑块的模式和分离

期刊:Development

影响因子:3.7

doi:10.1242/dev.183665

Li Wang, Junjie Xie, Haoran Zhang, Long Hin Tsang, Sze Lan Tsang, Eike-Benjamin Braune, Urban Lendahl, Mai Har Sham

Notch signaling promotes a HIF2α-driven hypoxic response in multiple tumor cell types

Notch 信号促进多种肿瘤细胞类型中 HIF2α 驱动的缺氧反应

期刊:Oncogene

影响因子:6.9

doi:10.1038/s41388-018-0400-3

Anders P Mutvei, Sebastian K-J Landor, Rhys Fox, Eike-Benjamin Braune, Yat Long Tsoi, Yee Peng Phoon, Cecilia Sahlgren, Johan Hartman, Jonas Bergh, Shaobo Jin, Urban Lendahl

Notch and Wnt Dysregulation and Its Relevance for Breast Cancer and Tumor Initiation

Notch和Wnt信号通路失调及其与乳腺癌和肿瘤发生的相关性

期刊:Biomedicines

影响因子:3.9

doi:10.3390/biomedicines6040101

Braune, Eike-Benjamin; Seshire, Anita; Lendahl, Urban

Notch

发表日期：2018

文献求助收藏

Loss of CSL Unlocks a Hypoxic Response and Enhanced Tumor Growth Potential in Breast Cancer Cells

CSL 的缺失会引发乳腺癌细胞的缺氧反应并增强肿瘤生长潜力

期刊:Stem Cell Reports

影响因子:5.9

doi:10.1016/j.stemcr.2016.03.004

Eike-Benjamin Braune, Yat Long Tsoi, Yee Peng Phoon, Sebastian Landor, Helena Silva Cascales, Daniel Ramsköld, Qiaolin Deng, Arne Lindqvist, Xiaojun Lian, Cecilia Sahlgren, Shao-Bo Jin, Urban Lendahl

S/T phosphorylation of DLL1 is required for full ligand activity in vitro but dispensable for DLL1 function in vivo during embryonic patterning and marginal zone B cell development

DLL1 的 S/T 磷酸化是体外完全配体活性所必需的，但对于胚胎模式形成和边缘区 B 细胞发育过程中的 DLL1 体内功能而言并非必需

期刊:Molecular and Cellular Biology

影响因子:3.2

doi:10.1128/MCB.00965-13

Eike-Benjamin Braune, Karin Schuster-Gossler, Marcin Lyszkiewicz, Katrin Serth, Kristina Preusse, Johannes Madlung, Boris Macek, Andreas Krueger, Achim Gossler

Identification of a Notch transcriptomic signature for breast cancer

乳腺癌 Notch 转录组特征的鉴定

Left ventricular hypertrophy and metabolic resetting in the Notch3-deficient adult mouse heart

Notch3 缺陷成年小鼠心脏左心室肥大和代谢重置

Roles of Notch Signaling in the Tumor Microenvironment

Notch信号通路在肿瘤微环境中的作用

Novel Cysteine-Sparing Hypomorphic NOTCH3 A1604T Mutation Observed in a Family With Migraine and White Matter Lesions

在患有偏头痛和白质病变的家族中发现了一种新型的保留半胱氨酸的低活性NOTCH3 A1604T突变

Notch signalling regulates epibranchial placode patterning and segregation

Notch 信号调节鳃上斑块的模式和分离

Notch signaling promotes a HIF2α-driven hypoxic response in multiple tumor cell types

Notch 信号促进多种肿瘤细胞类型中 HIF2α 驱动的缺氧反应

Notch and Wnt Dysregulation and Its Relevance for Breast Cancer and Tumor Initiation

Notch和Wnt信号通路失调及其与乳腺癌和肿瘤发生的相关性

Loss of CSL Unlocks a Hypoxic Response and Enhanced Tumor Growth Potential in Breast Cancer Cells

CSL 的缺失会引发乳腺癌细胞的缺氧反应并增强肿瘤生长潜力

S/T phosphorylation of DLL1 is required for full ligand activity in vitro but dispensable for DLL1 function in vivo during embryonic patterning and marginal zone B cell development

DLL1 的 S/T 磷酸化是体外完全配体活性所必需的，但对于胚胎模式形成和边缘区 B 细胞发育过程中的 DLL1 体内功能而言并非必需