H11 kinase/heat shock protein 22 deletion impairs both nuclear and mitochondrial functions of STAT3 and accelerates the transition into heart failure on cardiac overload
H11 激酶/热休克蛋白 22 缺失会损害 STAT3 的核和线粒体功能，并加速心脏超负荷时向心力衰竭的转变
期刊:Circulation
影响因子:35.5
doi:10.1161/CIRCULATIONAHA.110.013847
Hongyu Qiu, Paulo Lizano, Lydie Laure, Xiangzhen Sui, Eman Rashed, Ji Yeon Park, Chull Hong, Shumin Gao, Eric Holle, Didier Morin, Sunil K Dhar, Thomas Wagner, Alain Berdeaux, Bin Tian, Stephen F Vatner, Christophe Depre
