Emmanuel Matas相关文献与解析，生知库 | 链接生命科学的每一步

Yan Zheng ，Aiyi Liu ，Zi-Jun Wang ，Qing Cao ，Wei Wang ，Lin Lin ，Kaijie Ma ，Freddy Zhang ，Jing Wei ，Emmanuel Matas ，Jia Cheng ，Guo-Jun Chen ，Xiaomin Wang ，Zhen Yan

阿尔茨海默症

神经科学

EHMT1

发表日期：2019

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Sex-dependent behavioral deficits and neuropathology in a maternal immune activation model of autism

自闭症母体免疫激活模型中的性别相关行为缺陷和神经病理学

期刊:Translational Psychiatry

影响因子:5.8

doi:10.1038/s41398-019-0457-y

Obelia Haida, Tareq Al Sagheer, Anais Balbous, Maureen Francheteau, Emmanuel Matas, Federico Soria, Pierre Olivier Fernagut, Mohamed Jaber

Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition

通过抑制组蛋白去乙酰化酶 (HDAC) 可以挽救自闭症 Shank3 缺陷小鼠模型中的社交缺陷

期刊:Nature Neuroscience

影响因子:21.2

doi:10.1038/s41593-018-0110-8

Luye Qin #, Kaijie Ma #, Zi-Jun Wang, Zihua Hu, Emmanuel Matas, Jing Wei, Zhen Yan

Motor Impairments Correlate with Social Deficits and Restricted Neuronal Loss in an Environmental Model of Autism

在自闭症的环境模型中，运动障碍与社交缺陷和限制性神经元丢失相关

期刊:International Journal of Neuropsychopharmacology

影响因子:4.5

doi:10.1093/ijnp/pyy043

Tareq Al Sagheer, Obelia Haida, Anais Balbous, Maureen Francheteau, Emmanuel Matas, Pierre-Olivier Fernagut, Mohamed Jaber

Histone Modification of Nedd4 Ubiquitin Ligase Controls the Loss of AMPA Receptors and Cognitive Impairment Induced by Repeated Stress

Nedd4 泛素连接酶的组蛋白修饰控制 AMPA 受体的丢失和重复应激引起的认知障碍

期刊:Journal of Neuroscience

影响因子:4.4

doi:10.1523/JNEUROSCI.3056-15.2016

Jing Wei, Zhe Xiong, Janine B Lee, Jia Cheng, Lara J Duffney, Emmanuel Matas, Zhen Yan

Major motor and gait deficits with sexual dimorphism in a Shank3 mutant mouse model

Shank3 突变小鼠模型中存在主要运动和步态缺陷以及性别二态性

Inhibition of EHMT1/2 rescues synaptic and cognitive functions for Alzheimer's disease

抑制 EHMT1/2 可挽救阿尔茨海默病患者的突触和认知功能

Sex-dependent behavioral deficits and neuropathology in a maternal immune activation model of autism

自闭症母体免疫激活模型中的性别相关行为缺陷和神经病理学

Social deficits in Shank3-deficient mouse models of autism are rescued by histone deacetylase (HDAC) inhibition

通过抑制组蛋白去乙酰化酶 (HDAC) 可以挽救自闭症 Shank3 缺陷小鼠模型中的社交缺陷

Motor Impairments Correlate with Social Deficits and Restricted Neuronal Loss in an Environmental Model of Autism

在自闭症的环境模型中，运动障碍与社交缺陷和限制性神经元丢失相关

Histone Modification of Nedd4 Ubiquitin Ligase Controls the Loss of AMPA Receptors and Cognitive Impairment Induced by Repeated Stress

Nedd4 泛素连接酶的组蛋白修饰控制 AMPA 受体的丢失和重复应激引起的认知障碍