文献库，生知库 | 链接生命科学的每一步

日期影响因子

Association of CD4+ CD25+ T cells with prevention of severe destructive arthritis in Borrelia burgdorferi-vaccinated and challenged gamma interferon-deficient mice treated with anti-interleukin-17 antibody.

CD4+ CD25+ T 细胞与预防伯氏疏螺旋体疫苗接种和攻击的γ干扰素缺陷小鼠发生严重破坏性关节炎有关，这些小鼠接受了抗白介素-17抗体治疗

doi:10.1128/CDLI.11.6.1075-1084.2004

Nardelli Dean T, Burchill Matthew A, England Douglas M, Torrealba Jose, Callister Steven M, Schell Ronald F

细胞生物学

发表日期：2004

文献求助收藏

Neutralization of gamma interferon augments borreliacidal antibody production and severe destructive Lyme arthritis in C3H/HeJ mice.

γ干扰素的中和作用可增强C3H/HeJ小鼠体内抗伯氏疏螺旋体抗体的产生，并导致严重的破坏性莱姆关节炎

doi:10.1128/cdli.11.1.35-41.2004

Munson Erik L, DeCoster David J, Nardelli Dean T, England Douglas M, Callister Steven M, Schell Ronald F

发表日期：2004

文献求助收藏

Inhibition of interleukin-17 prevents the development of arthritis in vaccinated mice challenged with Borrelia burgdorferi.

抑制白细胞介素-17可防止接种疫苗的小鼠在感染伯氏疏螺旋体后发生关节炎

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.71.6.3437-3442.2003

Burchill Matthew A, Nardelli Dean T, England Douglas M, DeCoster David J, Christopherson John A, Callister Steven M, Schell Ronald F

细胞生物学

发表日期：2003

文献求助收藏

Destructive arthritis in vaccinated interferon gamma-deficient mice challenged with Borrelia burgdorferi: modulation by tumor necrosis factor alpha.

接种疫苗的干扰素γ缺乏小鼠感染伯氏疏螺旋体后发生破坏性关节炎：肿瘤坏死因子α的调节作用

doi:10.1128/cdli.10.1.44-52.2003

Christopherson John A, Munson Erik L, England Douglas M, Croke Cindy L, Remington Monica C, Molitor Melanie L, DeCoster David J, Callister Steven M, Schell Ronald F

发表日期：2003

文献求助收藏