日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Prothrombotic Transformation of the Endothelium Is Markedly Augmented By Cytokine Priming and Inhibited By Tie2 Activation

细胞因子预处理可显著增强内皮细胞的促血栓形成转化,而Tie2激活则可抑制该转化。

Liu, Michael; Wadhera, Rishi K; Eaton, Nathan; Enjyoji, Keiichi; Higgins, Sarah J; Doroudian, Maneli; Liao, Xinyuan; Flaumenhaft, Robert

Vasculopathy in COVID-19

新冠肺炎中的血管病变

Flaumenhaft, Robert; Enjyoji, Keiichi; Schmaier, Alec A

Structural and functional characterization of engineered bifunctional fusion proteins of CD39 and CD73 ectonucleotidases

CD39和CD73外核苷酸酶双功能融合蛋白的结构和功能表征

Zhong, Elizabeth H; Ledderose, Carola; De Andrade Mello, Paola; Enjyoji, Keiichi; Lunderberg, Justin Mark; Junger, Wolfgang; Robson, Simon C

Variation in glucose homeostasis traits associated with P2RX7 polymorphisms in mice and humans

小鼠和人类中与P2RX7多态性相关的葡萄糖稳态特征的变异

Todd, Jennifer N; Poon, Wenny; Lyssenko, Valeriya; Groop, Leif; Nichols, Brendan; Wilmot, Michael; Robson, Simon; Enjyoji, Keiichi; Herman, Mark A; Hu, Cheng; Zhang, Rong; Jia, Weiping; Ma, Ronald; Florez, Jose C; Friedman, David J

CD39 improves survival in microbial sepsis by attenuating systemic inflammation.

CD39 通过减轻全身炎症来提高微生物败血症的存活率

Csóka Balázs, Németh Zoltán H, Törő Gábor, Koscsó Balázs, Kókai Endre, Robson Simon C, Enjyoji Keiichi, Rolandelli Rolando H, Erdélyi Katalin, Pacher Pál, Haskó György

Characterization of circulating microparticle-associated CD39 family ecto-nucleotidases in human plasma

人血浆中循环微粒相关CD39家族胞外核苷酸酶的特征分析

Jiang, Z Gordon; Wu, Yan; Csizmadia, Eva; Feldbrügge, Linda; Enjyoji, Keiichi; Tigges, John; Toxavidis, Vasilis; Stephan, Holger; Müller, Christa E; McKnight, C James; Moss, Alan; Robson, Simon C

Potential factors influencing the development of thrombocytopenia and consumptive coagulopathy after genetically modified pig liver xenotransplantation

影响基因修饰猪肝异种移植后血小板减少症和消耗性凝血病发生的潜在因素

Ekser, Burcin; Lin, Chih C; Long, Cassandra; Echeverri, Gabriel J; Hara, Hidetaka; Ezzelarab, Mohamed; Bogdanov, Vladimir Y; Stolz, Donna B; Enjyoji, Keiichi; Robson, Simon C; Ayares, David; Dorling, Anthony; Cooper, David K C; Gridelli, Bruno

Liver damage and systemic inflammatory responses are exacerbated by the genetic deletion of CD39 in total hepatic ischemia.

肝脏完全缺血时,CD39 基因缺失会加剧肝损伤和全身炎症反应

Sun Xiaofeng, Imai Masato, Nowak-Machen Martina, Guckelberger Olaf, Enjyoji Keiichi, Wu Yan, Khalpey Zain, Berberat Pascal, Munasinghe Jeeva, Robson Simon Christopher

Deletion of CD39 on natural killer cells attenuates hepatic ischemia/reperfusion injury in mice

小鼠自然杀伤细胞上CD39的缺失可减轻肝脏缺血/再灌注损伤。

Beldi, Guido; Banz, Yara; Kroemer, Alexander; Sun, Xiaofeng; Wu, Yan; Graubardt, Nadine; Rellstab, Alyssa; Nowak, Martina; Enjyoji, Keiichi; Li, Xian; Junger, Wolfgang G; Candinas, Daniel; Robson, Simon C

From the Cover: CD39 deletion exacerbates experimental murine colitis and human polymorphisms increase susceptibility to inflammatory bowel disease

封面文章:CD39缺失会加剧实验性小鼠结肠炎,而人类多态性会增加炎症性肠病的易感性

Friedman, David J; Künzli, Beat M; A-Rahim, Yousif I; Sevigny, Jean; Berberat, Pascal O; Enjyoji, Keiichi; Csizmadia, Eva; Friess, Helmut; Robson, Simon C