H3F3A K27M mutations drive a repressive transcriptome by modulating chromatin accessibility independent of H3K27me3 in Diffuse Midline Glioma.
在弥漫性中线胶质瘤中，H3F3A K27M 突变通过调节染色质可及性驱动抑制性转录组，而这种调节与 H3K27me3 无关
期刊:Epigenetics & Chromatin
影响因子:3.5
doi:10.1186/s13072-025-00585-7
Bhattarai Suraj, Hakkim Faruck L, Day Charles A, Grigore Florina, Langfald Alyssa, Entin Igor, Hinchcliffe Edward H, Robinson James P
