日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Multiple modes of AFM reveal distinct mechanical properties for dystrophin and utrophin not manifest by small fragments

原子力显微镜的多种模式揭示了肌营养不良蛋白和肌营养蛋白各自独特的力学特性,而这些特性在小片段中是无法体现的。

Hua, Cailong; Vavra, Joseph; Powers, Jacob; Muretta, Joseph M; Ervasti, James M; Salapaka, Murti V

Proteomics-based evaluation of AAV dystrophin gene therapy outcomes in mdx skeletal muscle

基于蛋白质组学的AAV肌营养不良蛋白基因治疗在mdx骨骼肌中的疗效评估

Johnson, Erynn E; Reyes, Theodore R; Chamberlain, Jeffrey S; Ervasti, James M; Tasfaout, Hichem

The atomic structure of human dystrophin spectrin-like repeat 24

人类肌营养不良蛋白谱蛋白样重复序列24的原子结构

Streeter, Oakley; Shi, Ke; Bui, Hannah; Aihara, Hideki; Ervasti, James M; Evans, Robert 3rd; Muretta, Joseph M

Do actin isoforms have unique functionalities at the protein level?

肌动蛋白亚型在蛋白质水平上是否具有独特的功能?

Ervasti, James M; Kashina, Anna S; Perrin, Benjamin J

Human dystrophin tandem calponin homology actin-binding domain crystallized in a closed-state conformation

人类肌营养不良蛋白串联钙调蛋白同源肌动蛋白结合域以闭合状态构象结晶

Streeter, Oakley; Shi, Ke; Vavra, Joseph; Aihara, Hideki; Ervasti, James M; Evans, Robert 3rd; Muretta, Joseph M

Stress exposure in the mdx mouse model of Duchenne muscular dystrophy provokes a widespread metabolic response

在杜氏肌营养不良症的mdx小鼠模型中,应激暴露会引发广泛的代谢反应。

Johnson, Erynn E; Ervasti, James M

Fibroblast fate determination during cardiac reprogramming by remodeling of actin filaments.

心肌细胞重编程过程中,成纤维细胞的命运通过肌动蛋白丝的重塑而决定

Zhang Zhentao, Zhang Wenhui, Blakes Robert, Sundby Lauren J, Shi Zengdun, Rockey Don C, Ervasti James M, Nam Young-Jae

Stem Cell-Derived Cardiomyocytes and Beta-Adrenergic Receptor Blockade in Duchenne Muscular Dystrophy Cardiomyopathy

干细胞衍生心肌细胞和β-肾上腺素能受体阻滞剂治疗杜氏肌营养不良症心肌病

Kamdar, Forum; Das, Satyabrata; Gong, Wuming; Klaassen Kamdar, André; Meyers, Tatyana A; Shah, Pruthvi; Ervasti, James M; Townsend, DeWayne; Kamp, Timothy J; Wu, Joseph C; Garry, Mary G; Zhang, Jianyi; Garry, Daniel J

PFKFB3-mediated glycolysis rescues myopathic outcomes in the ischemic limb

PFKFB3介导的糖酵解可改善缺血肢体的肌病后果

Ryan, Terence E; Schmidt, Cameron A; Tarpey, Michael D; Amorese, Adam J; Yamaguchi, Dean J; Goldberg, Emma J; Iñigo, Melissa Mr; Karnekar, Reema; O'Rourke, Allison; Ervasti, James M; Brophy, Patricia; Green, Thomas D; Neufer, P Darrell; Fisher-Wellman, Kelsey; Spangenburg, Espen E; McClung, Joseph M

Loss of β-Cytoplasmic Actin in the Intestinal Epithelium Increases Gut Barrier Permeability in vivo and Exaggerates the Severity of Experimental Colitis.

肠上皮细胞中β-细胞质肌动蛋白的缺失会增加体内肠道屏障的通透性,并加剧实验性结肠炎的严重程度

Lechuga Susana, Naydenov Nayden G, Feygin Alex, Cruise Michael, Ervasti James M, Ivanov Andrei I