日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Real-time imaging of axonal membrane protein life cycles

轴突膜蛋白生命周期的实时成像

Tyagi, Sidharth; Higerd-Rusli, Grant P; Akin, Elizabeth J; Baker, Christopher A; Liu, Shujun; Dib-Hajj, Fadia B; Waxman, Stephen G; Dib-Hajj, Sulayman D

Inflammation differentially controls transport of depolarizing Nav versus hyperpolarizing Kv channels to drive rat nociceptor activity

炎症通过差异性地调控去极化Nav通道和超极化Kv通道的转运来驱动大鼠伤害感受器活性。

Higerd-Rusli, Grant P; Tyagi, Sidharth; Baker, Christopher A; Liu, Shujun; Dib-Hajj, Fadia B; Dib-Hajj, Sulayman D; Waxman, Stephen G

The fates of internalized NaV1.7 channels in sensory neurons: Retrograde cotransport with other ion channels, axon-specific recycling, and degradation

感觉神经元中内化的 NaV1.7 通道的命运:与其他离子通道的逆向共转运、轴突特异性循环和降解

Grant P Higerd-Rusli, Sidharth Tyagi, Shujun Liu, Fadia B Dib-Hajj, Stephen G Waxman, Sulayman D Dib-Hajj

Paclitaxel effects on axonal localization and vesicular trafficking of Na(V)1.8

紫杉醇对Na(V)1.8轴突定位和囊泡运输的影响

Baker, Christopher A; Tyagi, Sidharth; Higerd-Rusli, Grant P; Liu, Shujun; Zhao, Peng; Dib-Hajj, Fadia B; Waxman, Stephen G; Dib-Hajj, Sulayman D

Conserved but not critical: Trafficking and function of Na(V)1.7 are independent of highly conserved polybasic motifs

保守但不关键:Na(V)1.7 的转运和功能与高度保守的多碱性基序无关

Tyagi, Sidharth; Sarveswaran, Nivedita; Higerd-Rusli, Grant P; Liu, Shujun; Dib-Hajj, Fadia B; Waxman, Stephen G; Dib-Hajj, Sulayman D

Depolarizing NaV and Hyperpolarizing KV Channels Are Co-Trafficked in Sensory Neurons

去极化的 NaV 通道和超极化的 KV 通道在感觉神经元中共同流通

Grant P Higerd-Rusli, Matthew Alsaloum, Sidharth Tyagi, Nivedita Sarveswaran, Mark Estacion, Elizabeth J Akin, Fadia B Dib-Hajj, Shujun Liu, Daniel Sosniak, Peng Zhao, Sulayman D Dib-Hajj, Stephen G Waxman

Paclitaxel increases axonal localization and vesicular trafficking of Nav1.7

紫杉醇可增加 Nav1.7 的轴突定位和囊泡运输。

Akin, Elizabeth J; Alsaloum, Matthew; Higerd, Grant P; Liu, Shujun; Zhao, Peng; Dib-Hajj, Fadia B; Waxman, Stephen G; Dib-Hajj, Sulayman D

Lacosamide Inhibition of NaV1.7 Channels Depends on its Interaction With the Voltage Sensor Domain and the Channel Pore

拉科酰胺对 NaV1.7 通道的抑制取决于其与电压传感器域和通道孔的相互作用

Julie I R Labau, Matthew Alsaloum, Mark Estacion, Brian Tanaka, Fadia B Dib-Hajj, Giuseppe Lauria, Hubert J M Smeets, Catharina G Faber, Sulayman Dib-Hajj, Stephen G Waxman

KCNQ variants and pain modulation: a missense variant in Kv7.3 contributes to pain resilience

KCNQ 变异和疼痛调节:Kv7.3 中的错义变异有助于缓解疼痛

Jun-Hui Yuan, Mark Estacion, Malgorzata A Mis, Brian S Tanaka, Betsy R Schulman, Lubin Chen, Shujun Liu, Fadia B Dib-Hajj, Sulayman D Dib-Hajj, Stephen G Waxman

Building sensory axons: Delivery and distribution of NaV1.7 channels and effects of inflammatory mediators

构建感觉轴突:NaV1.7 通道的传递和分布以及炎症介质的影响

Elizabeth J Akin, Grant P Higerd-Rusli, Malgorzata A Mis, Brian S Tanaka, Talia Adi, Shujun Liu, Fadia B Dib-Hajj, Stephen G Waxman, Sulayman D Dib-Hajj