日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Stability of ARDS subphenotypes over time in two randomised controlled trials

两项随机对照试验中ARDS亚表型的稳定性

Delucchi, Kevin; Famous, Katie R; Ware, Lorraine B; Parsons, Polly E; Thompson, B Taylor; Calfee, Carolyn S

Acute Respiratory Distress Syndrome Subphenotypes Respond Differently to Randomized Fluid Management Strategy

急性呼吸窘迫综合征亚型对随机液体管理策略的反应不同

Famous, Katie R; Delucchi, Kevin; Ware, Lorraine B; Kangelaris, Kirsten N; Liu, Kathleen D; Thompson, B Taylor; Calfee, Carolyn S

Reply: Next Step to Understanding Subphenotypes of Acute Respiratory Distress Syndrome

回复:了解急性呼吸窘迫综合征亚表型的下一步

Calfee, Carolyn S; Delucchi, Kevin; Famous, Katie R

Muscarinic acetylcholine receptors in the nucleus accumbens core and shell contribute to cocaine priming-induced reinstatement of drug seeking

伏隔核核心和壳部的毒蕈碱型乙酰胆碱受体参与了可卡因启动诱导的觅药行为的复发。

Yee, Judy; Famous, Katie R; Hopkins, Thomas J; McMullen, Michael C; Pierce, R Christopher; Schmidt, Heath D

Cocaine-induced chromatin remodeling increases brain-derived neurotrophic factor transcription in the rat medial prefrontal cortex, which alters the reinforcing efficacy of cocaine.

可卡因诱导的染色质重塑增加了大鼠内侧前额叶皮层中脑源性神经营养因子的转录,从而改变了可卡因的强化效力

Sadri-Vakili Ghazaleh, Kumaresan Vidhya, Schmidt Heath D, Famous Katie R, Chawla Prianka, Vassoler Fair M, Overland Ryan P, Xia Eva, Bass Caroline E, Terwilliger Ernest F, Pierce R Christopher, Cha Jang-Ho J

The limbic circuitry underlying cocaine seeking encompasses the PPTg/LDT

可卡因渴求相关的边缘系统回路包含PPTg/LDT。

Schmidt, Heath D; Famous, Katie R; Pierce, R C

Metabotropic glutamate receptor 5 (mGluR5) antagonists attenuate cocaine priming- and cue-induced reinstatement of cocaine seeking

代谢型谷氨酸受体5 (mGluR5) 拮抗剂可减弱可卡因启动和线索诱导的可卡因渴求复发。

Kumaresan, Vidhya; Yuan, Menglu; Yee, Judy; Famous, Katie R; Anderson, Sharon M; Schmidt, Heath D; Pierce, R Christopher

When administered into the nucleus accumbens core or shell, the NMDA receptor antagonist AP-5 reinstates cocaine-seeking behavior in the rat

当将NMDA受体拮抗剂AP-5注射到大鼠伏隔核核心或壳层时,可恢复其对可卡因的渴求行为。

Famous, Katie R; Schmidt, Heath D; Pierce, R Christopher