日期 影响因子
日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:
–

Endothelial cell senescence shapes T cell activity in late-stage of chronic obstructive pulmonary disease

内皮细胞衰老影响慢性阻塞性肺疾病晚期T细胞的活性

期刊:Cell Death Discovery
影响因子:7
doi:10.1038/s41420-026-03020-2
Lee, Chae Min; Kim, Jongchan; Song, Junhyup; Kim, Andrew Sehoon; Jo, Sugyeong; Hwang, Nahee; Jeong, Jae Woong; Kim, Minki; Shin, Sung Jae; Fang, Sungsoon; Yoon, Bo Kyung
T细胞
慢性阻塞性肺疾病
细胞衰老
T细胞
衰老
内皮细胞
细胞生物学
免疫/内分泌
发表日期：2026
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Patient-Derived Organoids from Multiple Sites of a Single Tumor Recapitulates Intratumoral Heterogeneity in Patients with Gastric Cancer

源自同一肿瘤多个部位的患者来源类器官能够重现胃癌患者的肿瘤内异质性

期刊:Gut and Liver
影响因子:3.2
doi:10.5009/gnl250108
Yoon, Bo Kyung; Bae, Yoojin; Je, Yeonjin; Joo, Seyeon; Kim, Yuna; Shin, Su-Jin; Fang, Sungsoon; Kim, Jie-Hyun
胃癌
肿瘤
3D/类器官
肿瘤免疫
发表日期：2026
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FXR and BET signaling orchestrate to protect β cells

FXR和BET信号通路协同作用，保护β细胞

期刊:
影响因子:
doi:10.64898/2026.04.10.716420
Cayabyab, Fritz; Tipirneni, Jalan; Chen, David; Choi, Jinhyuk; Hamba, Yu; Pham, Nhi; Tacto, Clarissa; Wu, Jie; Wang, Liu; Mirzakhanyan, Yeva; Gershon, Paul David; Perez, Harvey; Harada, Naoki; Kim, Kiyoka; Shaheen, Ameen; Fang, Sungsoon; Ipp, Eli; Chen, Lin-Feng; Wei, Zong; Yoshihara, Eiji
信号转导
细胞生物学
发表日期：2026
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Bridging the gap: The GOLM1-OPN-ABCG5 axis in MASH and gallstone disease: Editorial on "GOLM1 promotes cholesterol gallstone formation via ABCG5-mediated cholesterol efflux in MASH livers"

弥合差距：GOLM1-OPN-ABCG5轴在MASH和胆结石疾病中的作用：关于“GOLM1通过ABCG5介导的胆固醇外流促进MASH肝脏中胆固醇胆结石的形成”的社论

期刊:Clinical and Molecular Hepatology
影响因子:16.9
doi:10.3350/cmh.2025.0066
Hwang, Nahee; Fang, Sungsoon
毒理研究
胆结石
代谢
发表日期：2025
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Reply to correspondence 2 on "GOLM1 promotes cholesterol gallstone formation via ABCG5-mediated cholesterol efflux in MASH livers"

回复关于“GOLM1通过ABCG5介导的胆固醇外流促进MASH肝脏中胆固醇结石的形成”的来信2

期刊:Clinical and Molecular Hepatology
影响因子:16.9
doi:10.3350/cmh.2025.0195
Hwang, Nahee; Fang, Sungsoon
代谢
发表日期：2025
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Author Correction: Novel mechanism whereby metformin improves glucose homeostasis: TXNIP-GLUT1 axis modulation enhances intestinal glucotonic effects

作者更正：二甲双胍改善葡萄糖稳态的新机制：TXNIP-GLUT1轴调节增强肠道葡萄糖张力作用

期刊:Experimental and Molecular Medicine
影响因子:12.9
doi:10.1038/s12276-025-01541-x
Kang, Chan Woo; Nam, Jung Ho; Oh, Ju Hun; Wang, Eun Kyung; Lee, Soohyun; Shin, Hye Ju; Kim, Ye Bin; Lee, Eun Jig; Lim, Byung Kook; Fang, Sungsoon; Cho, Arthur; Ku, Cheol Ryong
TXNIP
发表日期：2025
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SLC6A14-mediated glutamine promotes SYTL4-CXCL8 axis activation to drive gemcitabine resistance and immune evasion in pancreatic cancer.

SLC6A14 介导的谷氨酰胺促进 SYTL4-CXCL8 轴激活，从而驱动胰腺癌产生吉西他滨耐药性和免疫逃逸。

期刊:Experimental and Molecular Medicine
影响因子:12.9
doi:10.1038/s12276-025-01596-w
Kang Hyeon Woong, Kim Ju Hyun, Jeong Jae Woong, Fang Sungsoon, Yun Won-Gun, Jung Hye-Sol, Kwon Wooil, Jang Jin-Young, Kim Hyo Jung, Park Joon Seong
胰腺癌
免疫/内分泌
CXCL8
发表日期：2025
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PR inhibition stimulates G6PD expression to enhance malignancy in luminal breast cancer.

PR抑制可刺激G6PD表达，从而增强管腔型乳腺癌的恶性程度。

期刊:Cell Death & Disease
影响因子:9.6
doi:10.1038/s41419-025-08365-7
Jeong Jae Woong, Lee Janghee, Bae Soong June, Cha Yoon Jin, Fang Sungsoon, Lee Hae-Kyung, Ahn Sung Gwe
G6PD
肿瘤
肿瘤免疫
乳腺癌
发表日期：2025
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One-carbon metabolism is distinct metabolic signature for proliferative intermediate exhausted T cells of ICB-resistant cancer patients

单碳代谢是免疫检查点阻断耐药癌症患者增殖性中间耗竭T细胞的独特代谢特征。

期刊:Cell Death Discovery
影响因子:7
doi:10.1038/s41420-025-02332-z
Park, Ye-Chan; Hwang, Yeseong; Jeong, Jae Woong; Lee, Chae Min; Kim, Minki; Jo, Sugyeong; Joo, Seyeon; Hwang, Nahee; Fang, Sungsoon
肿瘤
T细胞
肿瘤免疫
代谢
T细胞
细胞生物学
免疫/内分泌
发表日期：2025
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One-carbon metabolic pathway is a novel molecular signature for CD44-positive intestinal-type gastric cancer

单碳代谢途径是CD44阳性肠型胃癌的一种新型分子标志。

期刊:Cell Death Discovery
影响因子:7
doi:10.1038/s41420-025-02704-5
Joo, Seyeon; Bae, Yoojin; Yoon, Bo Kyung; Je, Yeonjin; Kim, Yuna; Kim, Minki; Shin, Su-Jin; Fang, Sungsoon; Kim, Jie-Hyun
胃癌
肿瘤
T细胞
肿瘤免疫
代谢
CD44
CD4
发表日期：2025
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