日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Neurodevelopmental disorders: mechanisms and boundary definitions from genomes, interactomes and proteomes

神经发育障碍:来自基因组、相互作用组和蛋白质组的机制和边界定义

Mullin, A P; Gokhale, A; Moreno-De-Luca, A; Sanyal, S; Waddington, J L; Faundez, V

BLOC-1 complex deficiency alters the targeting of adaptor protein complex-3 cargoes

BLOC-1复合物缺陷会改变衔接蛋白复合物-3货物的靶向性

Salazar, G; Craige, B; Styers, M L; Newell-Litwa, K A; Doucette, M M; Wainer, B H; Falcon-Perez, J M; Dell'Angelica, E C; Peden, A A; Werner, E; Faundez, V

Genetic analysis of the neuronal and ubiquitous AP-3 adaptor complexes reveals divergent functions in brain

对神经元和普遍存在的AP-3衔接蛋白复合物的基因分析揭示了它们在大脑中的不同功能

Seong, E; Wainer, B H; Hughes, E D; Saunders, T L; Burmeister, M; Faundez, V

The neuronal form of adaptor protein-3 is required for synaptic vesicle formation from endosomes.

神经元形式的衔接蛋白-3是突触小泡从内体形成所必需的

Blumstein J, Faundez V, Nakatsu F, Saito T, Ohno H, Kelly R B

The AP-3 complex required for endosomal synaptic vesicle biogenesis is associated with a casein kinase Ialpha-like isoform.

内体突触小泡生物发生所需的 AP-3 复合物与酪蛋白激酶 Iα 样同工酶相关

Faundez V V, Kelly R B