日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

IL-1 protects from fatal systemic candidiasis in mice by inhibiting oxidative phosphorylation and hypoxia

IL-1通过抑制氧化磷酸化和缺氧来保护小鼠免受致命性全身性念珠菌病的侵害。

Sofia Horn,Mareike Schmid,Ivan Berest,Federica Piattini,Jing Zhang,Katrien de Bock,Olivier Devuyst,Stellor Nlandu Khodo,Jan Kisielow,Manfred Kopf

IL-1 protects from fatal systemic candidiasis in mice by inhibiting oxidative phosphorylation and hypoxia

IL-1通过抑制氧化磷酸化和缺氧来保护小鼠免受致命性全身性念珠菌病的侵害。

Sofia Horn ,Mareike Schmid ,Ivan Berest ,Federica Piattini ,Jing Zhang ,Katrien de Bock ,Olivier Devuyst ,Stellor Nlandu Khodo ,Jan Kisielow ,Manfred Kopf

Monocyte-derived alveolar macrophages autonomously determine severe outcome of respiratory viral infection

单核细胞衍生的肺泡巨噬细胞自主决定呼吸道病毒感染的严重后果

Fengqi Li, Federica Piattini, Lea Pohlmeier, Qian Feng, Hubert Rehrauer, Manfred Kopf

KappaBle fluorescent reporter mice enable low-background single-cell detection of NF-κB transcriptional activity in vivo

KappaBle 荧光报告小鼠可在体内低背景单细胞检测 NF-κB 转录活性

Luigi Tortola, Federica Piattini, Annika Hausmann, Franziska Ampenberger, Esther Rosenwald, Sebastian Heer, Wolf-Dietrich Hardt, Thomas Rülicke, Jan Kisielow, Manfred Kopf

Gene therapy of Csf2ra deficiency in mouse fetal monocyte precursors restores alveolar macrophage development and function

小鼠胎儿单核细胞前体细胞 Csf2ra 缺乏症的基因治疗可恢复肺泡巨噬细胞的发育和功能

Fengqi Li, Katarzyna Maria Okreglicka, Federica Piattini, Lea Maria Pohlmeier, Christoph Schneider, Manfred Kopf

Differential sensitivity of inflammatory macrophages and alternatively activated macrophages to ferroptosis

炎症巨噬细胞和替代活化巨噬细胞对铁死亡的不同敏感性

Federica Piattini, Mai Matsushita, Jonathan Muri, Peter Bretscher, Xiaogang Feng, Stefan Freigang, Jesmond Dalli, Christoph Schneider, Manfred Kopf

Differential sensitivity of inflammatory macrophages and alternatively activated macrophages to ferroptosis

炎症巨噬细胞和替代激活的巨噬细胞对铁死亡的敏感性存在差异

Federica Piattini,Mai Matsushita,Jonathan Muri,Peter Bretscher,Xiaogang Feng,Stefan Freigang,Jesmond Dalli,Christoph Schneider,Manfred Kopf

A spontaneous leptin receptor point mutation causes obesity and differentially affects leptin signaling in hypothalamic nuclei resulting in metabolic dysfunctions distinct from db/db mice

自发性瘦素受体点突变会导致肥胖,并对下丘脑核中的瘦素信号产生不同的影响,从而导致不同于 db/db 小鼠的代谢功能障碍

Federica Piattini, Christelle Le Foll, Jan Kisielow, Esther Rosenwald, Peter Nielsen, Thomas Lutz, Christoph Schneider, Manfred Kopf