Fernanda B M相关文献与解析，生知库 | 链接生命科学的每一步

Virulence genes of Rickettsia rickettsii are differentially modulated by either temperature upshift or blood-feeding in tick midgut and salivary glands

立克次体毒力基因在蜱虫中肠和唾液腺中会受到温度升高或吸血的不同程度的调节。

Galletti, Maria Fernanda B M; Fujita, André; Rosa, Rafael D; Martins, Larissa A; Soares, Herbert S; Labruna, Marcelo B; Daffre, Sirlei; Fogaça, Andréa C

发表日期：2016

文献求助收藏

Impaired Corpus Cavernosum Relaxation Is Accompanied by Increased Oxidative Stress and Up-Regulation of the Rho-Kinase Pathway in Diabetic (Db/Db) Mice

糖尿病 (Db/Db) 小鼠的阴茎海绵体松弛受损伴有氧化应激增加和 Rho-激酶通路上调

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0156030

Fernanda B M Priviero, Haroldo A F Toque, Kenia Pedrosa Nunes, Denise G Priolli, Cleber E Teixeira, R Clinton Webb

Natural blood feeding and temperature shift modulate the global transcriptional profile of Rickettsia rickettsii infecting its tick vector

自然吸血和温度变化会调节感染蜱虫媒介的立克次体的整体转录谱。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0077388

Galletti, Maria Fernanda B M; Fujita, André; Nishiyama, Milton Y Jr; Malossi, Camila D; Pinter, Adriano; Soares, João F; Daffre, Sirlei; Labruna, Marcelo B; Fogaça, Andréa C

炎症/感染

发表日期：2013

文献求助收藏

Up-regulation of the RhoA/Rho-kinase signaling pathway in corpus cavernosum from endothelial nitric-oxide synthase (NOS), but not neuronal NOS, null mice

内皮型一氧化氮合酶 (NOS) 缺失小鼠（而非神经元型 NOS 缺失小鼠）可上调阴茎海绵体中的 RhoA/Rho 激酶信号通路

期刊:Journal of Pharmacology and Experimental Therapeutics

影响因子:3.1

doi:10.1124/jpet.109.160606

Fernanda B M Priviero, Li-Ming Jin, Zhekang Ying, Cleber E Teixeira, R Clinton Webb

神经

发表日期：2010

文献求助收藏

Heme-dependent and independent soluble guanylate cyclase activators and vasodilation

血红素依赖性和非依赖性可溶性鸟苷酸环化酶激活剂及血管舒张作用

期刊:Journal of Cardiovascular Pharmacology

影响因子:2.2

doi:10.1097/FJC.0b013e3181eb4e75

Priviero, Fernanda B M; Webb, R Clinton

发表日期：2010

文献求助收藏

Oxidative stress impairs vasorelaxation induced by the soluble guanylyl cyclase activator BAY 41-2272 in spontaneously hypertensive rats

氧化应激损害自发性高血压大鼠中可溶性鸟苷酸环化酶激活剂 BAY 41-2272 诱导的血管舒张作用

期刊:American Journal of Hypertension

影响因子:3.2

doi:10.1038/ajh.2009.18

Fernanda B M Priviero, Saiprasad M Zemse, Cleber E Teixeira, R Clinton Webb

Constitutive LH receptor activity impairs NO-mediated penile smooth muscle relaxation

组成性 LH 受体活性损害 NO 介导的阴茎平滑肌松弛

Virulence genes of Rickettsia rickettsii are differentially modulated by either temperature upshift or blood-feeding in tick midgut and salivary glands

立克次体毒力基因在蜱虫中肠和唾液腺中会受到温度升高或吸血的不同程度的调节。

Impaired Corpus Cavernosum Relaxation Is Accompanied by Increased Oxidative Stress and Up-Regulation of the Rho-Kinase Pathway in Diabetic (Db/Db) Mice

糖尿病 (Db/Db) 小鼠的阴茎海绵体松弛受损伴有氧化应激增加和 Rho-激酶通路上调

Natural blood feeding and temperature shift modulate the global transcriptional profile of Rickettsia rickettsii infecting its tick vector

自然吸血和温度变化会调节感染蜱虫媒介的立克次体的整体转录谱。

Up-regulation of the RhoA/Rho-kinase signaling pathway in corpus cavernosum from endothelial nitric-oxide synthase (NOS), but not neuronal NOS, null mice

内皮型一氧化氮合酶 (NOS) 缺失小鼠（而非神经元型 NOS 缺失小鼠）可上调阴茎海绵体中的 RhoA/Rho 激酶信号通路

Heme-dependent and independent soluble guanylate cyclase activators and vasodilation

血红素依赖性和非依赖性可溶性鸟苷酸环化酶激活剂及血管舒张作用

Oxidative stress impairs vasorelaxation induced by the soluble guanylyl cyclase activator BAY 41-2272 in spontaneously hypertensive rats

氧化应激损害自发性高血压大鼠中可溶性鸟苷酸环化酶激活剂 BAY 41-2272 诱导的血管舒张作用