Overexpression of alpha synuclein disrupts APP and Endolysosomal axonal trafficking in a mouse model of synucleinopathy
在突触核蛋白病小鼠模型中，α-突触核蛋白的过度表达会破坏APP和内溶酶体轴突运输。
期刊:Neurobiology of Disease
影响因子:5.6
doi:10.1016/j.nbd.2023.106010
Lin, Suzhen; Leitão, André D G; Fang, Savannah; Gu, Yingli; Barber, Sophia; Gilliard-Telefoni, Rhiannon; Castro, Alfredo; Sung, Kijung; Shen, Ruinan; Florio, Jazmin B; Mante, Michael L; Ding, Jianqing; Spencer, Brian; Masliah, Eliezer; Rissman, Robert A; Wu, Chengbiao
