Fonseca Maria I相关文献与解析，生知库 | 链接生命科学的每一步

Complement activation fragment C5a receptors, CD88 and C5L2, are associated with neurofibrillary pathology.

补体激活片段 C5a 受体、CD88 和 C5L2 与神经纤维缠结病理有关

期刊:Journal of Neuroinflammation

影响因子:10.1

doi:10.1186/1742-2094-10-25

Fonseca Maria I, McGuire Susan O, Counts Scott E, Tenner Andrea J

Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease

补体激活途径对阿尔茨海默病小鼠模型神经病理学的贡献各不相同

期刊:Journal of Neuroinflammation

影响因子:

doi:10.1186/1742-2094-8-4

Fonseca, Maria I; Chu, Shu-Hui; Berci, Alisia M; Benoit, Marie E; Peters, Douglas G; Kimura, Yuko; Tenner, Andrea J

Treatment with a C5aR antagonist decreases pathology and enhances behavioral performance in murine models of Alzheimer's disease

使用C5aR拮抗剂治疗可减轻阿尔茨海默病小鼠模型的病理变化并改善其行为表现。

期刊:Journal of Immunology

影响因子:

doi:10.4049/jimmunol.0901005

Fonseca, Maria I; Ager, Rahasson R; Chu, Shu-Hui; Yazan, Ozkan; Sanderson, Sam D; LaFerla, Frank M; Taylor, Stephen M; Woodruff, Trent M; Tenner, Andrea J

阿尔茨海默症

发表日期：2009

文献求助收藏

Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease

阿尔茨海默病C1q充足和缺陷小鼠模型中补体C3和C4的表达

期刊:Journal of Neurochemistry

影响因子:4

doi:10.1111/j.1471-4159.2008.05558.x

Zhou, Jun; Fonseca, Maria I; Pisalyaput, Karntipa; Tenner, Andrea J

Novel Abeta peptide immunogens modulate plaque pathology and inflammation in a murine model of Alzheimer's disease

新型Aβ肽免疫原可调节阿尔茨海默病小鼠模型中的斑块病理和炎症

期刊:Journal of Neuroinflammation

影响因子:10.1

doi:10.1186/1742-2094-2-28

Zhou, Jun; Fonseca, Maria I; Kayed, Rakez; Hernandez, Irma; Webster, Scott D; Yazan, Ozkan; Cribbs, David H; Glabe, Charles G; Tenner, Andrea J

C5a Increases the Injury to Primary Neurons Elicited by Fibrillar Amyloid Beta

C5a 会加剧纤维状β淀粉样蛋白对原代神经元的损伤。

Complement activation fragment C5a receptors, CD88 and C5L2, are associated with neurofibrillary pathology.

补体激活片段 C5a 受体、CD88 和 C5L2 与神经纤维缠结病理有关

Contribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease

补体激活途径对阿尔茨海默病小鼠模型神经病理学的贡献各不相同

Treatment with a C5aR antagonist decreases pathology and enhances behavioral performance in murine models of Alzheimer's disease

使用C5aR拮抗剂治疗可减轻阿尔茨海默病小鼠模型的病理变化并改善其行为表现。

Complement C3 and C4 expression in C1q sufficient and deficient mouse models of Alzheimer's disease

阿尔茨海默病C1q充足和缺陷小鼠模型中补体C3和C4的表达

Novel Abeta peptide immunogens modulate plaque pathology and inflammation in a murine model of Alzheimer's disease

新型Aβ肽免疫原可调节阿尔茨海默病小鼠模型中的斑块病理和炎症