Fontes Milene T相关文献与解析，生知库 | 链接生命科学的每一步

Fontes, Milene T; Townsend, Paul; Butler, Landon; Parente, Juliana M; Araujo, Fênix A; Costa, Tiago J; Bomfim, Gisele F; Pernomian, Laena; Tan, Wenbin; VanderVeen, Brandon N; Murphy, E Angela; Stark, Ryan J; Butcher, Joshua T; McCarthy, Cameron G; Wenceslau, Camilla F

心血管

高血压

发表日期：2026

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The loss of glycoprotein nonmetastatic melanoma protein B (GPNMB) alters endothelial cell permeability, metabolism, and survival during infectious challenge

非转移性黑色素瘤糖蛋白B (GPNMB) 的缺失会改变内皮细胞的通透性、代谢以及在感染挑战下的存活率。

期刊:Clinical Science

影响因子:7.7

doi:10.1042/CS20256682

Fontes, Milene T; Lamb, Celia K; Pourquoi, Allison; Atzrodt, Cassandra Lauren; Nguyen, Hong-Ngan; Panja, Sourav; Stark, Ryan Jordan

Overexpression of adipose tissue ERÎ± enhances PVAT anticontractility via NOX4-derived H(2)O(2) and is protective against high-fat diet-induced dysfunction.

脂肪组织 ERα 的过度表达通过 NOX4 衍生的 H(2)O(2) 增强 PVAT 的抗收缩性，并能保护 PVAT 免受高脂饮食引起的机能障碍的影响

期刊:American Journal of Physiology-Heart and Circulatory Physiology

影响因子:4.1

doi:10.1152/ajpheart.00180.2025

Costa Tiago J, Fontes Milene T, Barros Paula R, Hope Marion C, Webb R Clinton, Wenceslau Camilla F, Enos Reilly T, McCarthy Cameron G

NOX4

发表日期：2025

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A Single-Short Partial Reprogramming of the Endothelial Cells decreases Blood Pressure via attenuation of EndMT in Hypertensive Mice

对内皮细胞进行单次短期部分重编程可通过减弱高血压小鼠的内皮间质转化（EndMT）来降低血压。

期刊:

影响因子:

doi:10.1101/2024.05.20.595057

Pernomian, Laena; Waigi, Emily W; Nguyen, Vi; Mohammed, Ahmed D; da Costa, Tiago J; Fontes, Milene T; Kubinak, Jason L; Aitken, Andrew; Biancardi, Vinicia Campana; Sinclair, David A; McCarthy, Cameron G; Wang, Yunguan; Tan, Wenbin; Wenceslau, Camilla Ferreira

The O-GlcNAc dichotomy: when does adaptation become pathological?

O-GlcNAc 二分法：适应何时会变成病态？

期刊:Clinical Science

影响因子:7.7

doi:10.1042/CS20220309

Costa, Tiago J; Wilson, Emily W; Fontes, Milene T; Pernomian, Laena; Tostes, Rita C; Wenceslau, Camilla F; McCarthy, Cameron G

发表日期：2023

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Specialized Pro-resolving Mediator Improves Vascular Relaxation via Formyl Peptide Receptor-2

特异性促消退介质通过甲酰肽受体-2改善血管舒张

期刊:American Journal of Hypertension

影响因子:3.1

doi:10.1093/ajh/hpad062

Edwards-Glenn, Jonnelle M; Fontes, Milene T; Waigi, Emily W; Costa, Tiago J; Maiseyeu, Andrei; Webb, R Clinton; McCarthy, Cameron G; Wenceslau, Camilla F

信号转导

发表日期：2023

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Hypertension Drives Protein Lactylation and Vascular Dysfunction in Skeletal Muscle

高血压导致骨骼肌蛋白质乳化和血管功能障碍

The loss of glycoprotein nonmetastatic melanoma protein B (GPNMB) alters endothelial cell permeability, metabolism, and survival during infectious challenge

非转移性黑色素瘤糖蛋白B (GPNMB) 的缺失会改变内皮细胞的通透性、代谢以及在感染挑战下的存活率。

Overexpression of adipose tissue ERÎ± enhances PVAT anticontractility via NOX4-derived H(2)O(2) and is protective against high-fat diet-induced dysfunction.

脂肪组织 ERα 的过度表达通过 NOX4 衍生的 H(2)O(2) 增强 PVAT 的抗收缩性，并能保护 PVAT 免受高脂饮食引起的机能障碍的影响

A Single-Short Partial Reprogramming of the Endothelial Cells decreases Blood Pressure via attenuation of EndMT in Hypertensive Mice

对内皮细胞进行单次短期部分重编程可通过减弱高血压小鼠的内皮间质转化（EndMT）来降低血压。

The O-GlcNAc dichotomy: when does adaptation become pathological?

O-GlcNAc 二分法：适应何时会变成病态？

Specialized Pro-resolving Mediator Improves Vascular Relaxation via Formyl Peptide Receptor-2

特异性促消退介质通过甲酰肽受体-2改善血管舒张