日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

GLI1+ perivascular, renal, progenitor cells: The likely source of spontaneous neoplasia that created the AGMK1-9T7 cell line

GLI1+血管周围肾脏祖细胞:可能是自发性肿瘤形成AGMK1-9T7细胞系的来源

Andrew M Lewis Jr ,Gideon Foseh ,Wei Tu ,Keith Peden ,Adovi Akue ,Mark KuKuruga ,Daniel Rotroff ,Gladys Lewis ,Ilya Mazo ,Steven R Bauer

The AGMK1-9T7 cell model of neoplasia: Evolution of DNA copy-number aberrations and miRNA expression during transition from normal to metastatic cancer cells

AGMK1-9T7细胞肿瘤模型:从正常细胞向转移性癌细胞转变过程中DNA拷贝数异常和miRNA表达的演变

Lewis, Andrew M Jr; Thomas, Rachael; Breen, Matthew; Peden, Keith; Teferedegne, Belete; Foseh, Gideon; Motsinger-Reif, Alison; Rotroff, Daniel; Lewis, Gladys

Responsiveness to basement membrane extract as a possible trait for tumorigenicity characterization

对基底膜提取物的反应性作为肿瘤发生特性的一种可能特征

Murata, Haruhiko; Omeir, Romelda; Tu, Wei; Lanning, Lynda; Phy, Kathryn; Foseh, Gideon; Lewis, Andrew M Jr; Peden, Keith

A novel canine kidney cell line model for the evaluation of neoplastic development: karyotype evolution associated with spontaneous immortalization and tumorigenicity

用于评估肿瘤发展的新型犬肾细胞系模型:与自发永生化和致瘤性相关的核型进化

R Omeir, R Thomas, B Teferedegne, C Williams, G Foseh, J Macauley, L Brinster, J Beren, K Peden, M Breen, A M Lewis Jr0

A mouse strain defective in both T cells and NK cells has enhanced sensitivity to tumor induction by plasmid DNA expressing both activated H-Ras and c-Myc.

T 细胞和 NK 细胞均有缺陷的小鼠品系对表达活化 H-Ras 和 c-Myc 的质粒 DNA 诱导肿瘤具有更高的敏感性

Sheng-Fowler Li, Tu Wei, Fu Haiqing, Murata Haruhiko, Lanning Lynda, Foseh Gideon, Macauley Juliete, Blair Donald, Hughes Stephen H, Coffin John M, Lewis Andrew M Jr, Peden Keith

Tumors induced in mice by direct inoculation of plasmid DNA expressing both activated H-ras and c-myc

通过直接接种表达活化H-ras和c-myc的质粒DNA,在小鼠体内诱导肿瘤。

Sheng-Fowler, Li; Cai, Fang; Fu, Haiqing; Zhu, Yong; Orrison, Brian; Foseh, Gideon; Blair, Don G; Hughes, Stephen H; Coffin, John M; Lewis, Andrew M Jr; Peden, Keith