日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Autoreactive B cells recruited to lungs by silica exposure contribute to local autoantibody production in autoimmune-prone BXSB and B cell receptor transgenic mice

二氧化硅暴露后募集到肺部的自身反应性B细胞,促进了易患自身免疫性疾病的BXSB小鼠和B细胞受体转基因小鼠的局部自身抗体产生。

Fee, Lanette; Kumar, Advika; Tighe, Robert M; Foster, Mary H

Altered toll-like receptor responsiveness underlies a dominant heritable defect in B cell tolerance in autoimmune New Zealand Black mice

新西兰黑鼠自身免疫性疾病中,Toll样受体反应性的改变是B细胞耐受性显性遗传缺陷的根本原因。

Clark, Amy G; Buckley, Elizabeth S; Foster, Mary H

Basement membranes and autoimmune diseases

基底膜和自身免疫性疾病

Foster, Mary H

A murine Ig light chain transgene reveals IGKV3 gene contributions to anti-collagen types IV and II specificities

小鼠免疫球蛋白轻链转基因揭示了IGKV3基因对IV型和II型胶原蛋白特异性的贡献

Clark, Amy G; Worni-Schudel, Inge M; Korte, Francesca M; Foster, Mary H

Uncommon structural motifs dominate the antigen binding site in human autoantibodies reactive with basement membrane collagen

在与基底膜胶原蛋白反应的人类自身抗体中,抗原结合位点主要由不常见的结构基序构成。

Foster, Mary H; Buckley, Elizabeth S; Chen, Benny J; Hwang, Kwan-Ki; Clark, Amy G

Optimizing the translational value of animal models of glomerulonephritis: insights from recent murine prototypes

优化肾小球肾炎动物模型的转化价值:来自近期小鼠原型模型的启示

Foster, Mary H

Recovery of a human natural antibody against the noncollagenous-1 domain of type IV collagen using humanized models

利用人源化模型回收针对IV型胶原蛋白非胶原-1结构域的人类天然抗体

Worni-Schudel, Inge M; Clark, Amy G; Chien, Tiffany; Hwang, Kwan-Ki; Chen, Benny J; Foster, Mary H

Lack of galectin-1 or galectin-3 alters B cell deletion and anergy in an autoantibody transgene model

在自身抗体转基因模型中,半乳糖凝集素-1或半乳糖凝集素-3的缺失会改变B细胞的缺失和无反应性。

Clark, Amy G; Weston, Melissa L; Foster, Mary H

Regulation of basement membrane-reactive B cells in BXSB, (NZBxNZW)F1, NZB, and MRL/lpr lupus mice

BXSB、(NZBxNZW)F1、NZB 和 MRL/lpr 狼疮小鼠中基底膜反应性 B 细胞的调控

Clark, Amy G; Fan, Qihua; Brady, Graham F; Mackin, Katherine M; Coffman, Evan D; Weston, Melissa L; Foster, Mary H

Genetic elimination of α3(IV) collagen fails to rescue anti-collagen B cells.

基因消除α3(IV)胶原蛋白无法挽救抗胶原蛋白B细胞

Clark Amy G, Mackin Katherine M, Foster Mary H