日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

EpoR Activation Stimulates Erythroid Precursor Proliferation by Inducing Phosphorylation of Tyrosine-88 of the CDK-Inhibitor p27Kip1

EpoR激活通过诱导CDK抑制剂p27Kip1的酪氨酸-88磷酸化来刺激红系前体细胞增殖

Fragka Pegka ,Nathalie Ben-Califa ,Drorit Neumann ,Heidelinde Jäkel ,Ludger Hengst

Inability to phosphorylate Y88 of p27(Kip1) enforces reduced p27 protein levels and accelerates leukemia progression

p27(Kip1)的Y88位点磷酸化能力不足会导致p27蛋白水平降低,并加速白血病进展。

Jäkel, Heidelinde; Taschler, Martin; Jung, Karin; Weinl, Christina; Pegka, Fragka; Kullmann, Michael Keith; Podmirseg, Silvio Roland; Dutta, Sayantanee; Moser, Markus; Hengst, Ludger

The SKP2-p27 axis defines susceptibility to cell death upon CHK1 inhibition

SKP2-p27 轴决定了 CHK1 抑制后细胞死亡的敏感性

Michael Lohmüller, Bernhard F Roeck, Tamas G Szabo, Marina A Schapfl, Fragka Pegka, Sebastian Herzog, Andreas Villunger, Fabian Schuler

Stimulation of c-Jun/AP-1-Activity by the Cell Cycle Inhibitor p57Kip2

细胞周期抑制剂 p57Kip2 刺激 c-Jun/AP-1 活性

Michael Keith Kullmann, Fragka Pegka, Christian Ploner, Ludger Hengst