Francesca Riuzzi相关文献与解析，生知库 | 链接生命科学的每一步

Giorgia Renga, Emilia Nunzi, Marilena Pariano, Matteo Puccetti, Marina Maria Bellet, Giuseppe Pieraccini, Fiorella D'Onofrio, Ilaria Santarelli, Claudia Stincardini, Franco Aversa, Francesca Riuzzi, Cinzia Antognelli, Marco Gargaro, Oxana Bereshchenko, Maurizio Ricci, Stefano Giovagnoli, Luigina Rom

代谢

免疫

Checkpoint

发表日期：2022

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Sertoli Cells Improve Myogenic Differentiation, Reduce Fibrogenic Markers, and Induce Utrophin Expression in Human DMD Myoblasts

塞托利细胞可促进成肌分化、降低纤维化标志物并诱导人类 DMD 成肌细胞中 Utrophin 的表达

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom11101504

Laura Salvadori, Sara Chiappalupi, Iva Arato, Francesca Mancuso, Mario Calvitti, Maria Cristina Marchetti, Francesca Riuzzi, Riccardo Calafiore, Giovanni Luca, Guglielmo Sorci

Microencapsulated Sertoli cells sustain myoblast proliferation without affecting the myogenic potential. In vitro data

微囊化塞托利细胞可维持成肌细胞增殖，而不会影响成肌潜能。体外数据

期刊:Data in Brief

影响因子:1

doi:10.1016/j.dib.2021.107744

Sara Chiappalupi, Laura Salvadori, Francesca Mancuso, Iva Arato, Mario Calvitti, Francesca Riuzzi, Riccardo Calafiore, Giovanni Luca, Guglielmo Sorci

Targeting RAGE prevents muscle wasting and prolongs survival in cancer cachexia

靶向 RAGE 可防止肌肉萎缩并延长癌症恶病质患者的生存期

期刊:Journal of Cachexia Sarcopenia and Muscle

影响因子:9.4

doi:10.1002/jcsm.12561

Sara Chiappalupi, Guglielmo Sorci, Aleksandra Vukasinovic, Laura Salvadori, Roberta Sagheddu, Dario Coletti, Giorgia Renga, Luigina Romani, Rosario Donato, Francesca Riuzzi

肿瘤

发表日期：2020

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Oxidative stress-induced S100B accumulation converts myoblasts into brown adipocytes via an NF-κB/YY1/miR-133 axis and NF-κB/YY1/BMP-7 axis

氧化应激诱导的 S100B 积累通过 NF-κB/YY1/miR-133 轴和 NF-κB/YY1/BMP-7 轴将成肌细胞转化为棕色脂肪细胞

期刊:Cell Death and Differentiation

影响因子:13.7

doi:10.1038/cdd.2017.132

Giulio Morozzi, Sara Beccafico, Roberta Bianchi, Francesca Riuzzi, Ilaria Bellezza, Ileana Giambanco, Cataldo Arcuri, Alba Minelli, Rosario Donato

Levels of S100B protein drive the reparative process in acute muscle injury and muscular dystrophy

S100B 蛋白水平推动急性肌肉损伤和肌营养不良症的修复过程

期刊:Scientific Reports

影响因子:3.8

doi:10.1038/s41598-017-12880-9

Francesca Riuzzi, Sara Beccafico, Roberta Sagheddu, Sara Chiappalupi, Ileana Giambanco, Oxana Bereshchenko, Carlo Riccardi, Guglielmo Sorci, Rosario Donato

信号转导

发表日期：2017

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RAGE signaling deficiency in rhabdomyosarcoma cells causes upregulation of PAX7 and uncontrolled proliferation

横纹肌肉瘤细胞中 RAGE 信号缺陷导致 PAX7 上调和不受控制的增殖

期刊:Journal of Cell Science

影响因子:3.3

doi:10.1242/jcs.136259

Francesca Riuzzi, Guglielmo Sorci, Roberta Sagheddu, Angelo Sidoni, Rita Alaggio, Vito Ninfo, Rosario Donato

Phosphocaveolin-1 enforces tumor growth and chemoresistance in rhabdomyosarcoma

磷酸化洞穴蛋白-1 促进横纹肌肉瘤的肿瘤生长和化学耐药性

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0084618

Fiorella Faggi, Stefania Mitola, Guglielmo Sorci, Francesca Riuzzi, Rosario Donato, Silvia Codenotti, Pietro Luigi Poliani, Manuela Cominelli, Raffaella Vescovi, Stefania Rossi, Stefano Calza, Marina Colombi, Fabio Penna, Paola Costelli, Ilaria Perini, Maurilio Sampaolesi, Eugenio Monti, Alessandro

HuR and miR-1192 regulate myogenesis by modulating the translation of HMGB1 mRNA

HuR 和 miR-1192 通过调节 HMGB1 mRNA 的翻译来调节肌肉生成

期刊:Nature Communications

影响因子:14.7

doi:10.1038/ncomms3388

Virginie Dormoy-Raclet, Anne Cammas, Barbara Celona, Xian Jin Lian, Kate van der Giessen, Marija Zivojnovic, Silvia Brunelli, Francesca Riuzzi, Guglielmo Sorci, Brian T Wilhelm, Sergio Di Marco, Rosario Donato, Marco E Bianchi, Imed-Eddine Gallouzi

HMGB1

发表日期：2013

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S100B engages RAGE or bFGF/FGFR1 in myoblasts depending on its own concentration and myoblast density. Implications for muscle regeneration

S100B 根据其自身浓度和成肌细胞密度与成肌细胞中的 RAGE 或 bFGF/FGFR1 结合。对肌肉再生的影响

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0028700

Francesca Riuzzi, Guglielmo Sorci, Sara Beccafico, Rosario Donato

Optimizing therapeutic outcomes of immune checkpoint blockade by a microbial tryptophan metabolite

通过微生物色氨酸代谢物优化免疫检查点阻断的治疗效果

Sertoli Cells Improve Myogenic Differentiation, Reduce Fibrogenic Markers, and Induce Utrophin Expression in Human DMD Myoblasts

塞托利细胞可促进成肌分化、降低纤维化标志物并诱导人类 DMD 成肌细胞中 Utrophin 的表达

Microencapsulated Sertoli cells sustain myoblast proliferation without affecting the myogenic potential. In vitro data

微囊化塞托利细胞可维持成肌细胞增殖，而不会影响成肌潜能。体外数据

Targeting RAGE prevents muscle wasting and prolongs survival in cancer cachexia

靶向 RAGE 可防止肌肉萎缩并延长癌症恶病质患者的生存期

Oxidative stress-induced S100B accumulation converts myoblasts into brown adipocytes via an NF-κB/YY1/miR-133 axis and NF-κB/YY1/BMP-7 axis

氧化应激诱导的 S100B 积累通过 NF-κB/YY1/miR-133 轴和 NF-κB/YY1/BMP-7 轴将成肌细胞转化为棕色脂肪细胞

Levels of S100B protein drive the reparative process in acute muscle injury and muscular dystrophy

S100B 蛋白水平推动急性肌肉损伤和肌营养不良症的修复过程

RAGE signaling deficiency in rhabdomyosarcoma cells causes upregulation of PAX7 and uncontrolled proliferation

横纹肌肉瘤细胞中 RAGE 信号缺陷导致 PAX7 上调和不受控制的增殖

Phosphocaveolin-1 enforces tumor growth and chemoresistance in rhabdomyosarcoma

磷酸化洞穴蛋白-1 促进横纹肌肉瘤的肿瘤生长和化学耐药性

HuR and miR-1192 regulate myogenesis by modulating the translation of HMGB1 mRNA

HuR 和 miR-1192 通过调节 HMGB1 mRNA 的翻译来调节肌肉生成

S100B engages RAGE or bFGF/FGFR1 in myoblasts depending on its own concentration and myoblast density. Implications for muscle regeneration

S100B 根据其自身浓度和成肌细胞密度与成肌细胞中的 RAGE 或 bFGF/FGFR1 结合。对肌肉再生的影响