Activation of invariant NKT cells ameliorates experimental ocular autoimmunity by a mechanism involving innate IFN-gamma production and dampening of the adaptive Th1 and Th17 responses
激活不变性NKT细胞可通过一种涉及先天性IFN-γ产生和抑制适应性Th1和Th17反应的机制,改善实验性眼部自身免疫性疾病。
期刊:Journal of Immunology
影响因子:3.4
doi:10.4049/jimmunol.181.7.4791
Grajewski, Rafael S; Hansen, Anna M; Agarwal, Rajeev K; Kronenberg, Mitchell; Sidobre, Stephane; Su, Shao Bo; Silver, Phyllis B; Tsuji, Moriya; Franck, Richard W; Lawton, Anne P; Chan, Chi-Chao; Caspi, Rachel R
T细胞
自身免疫性疾病
T细胞
IFN-γ
细胞生物学
免疫/内分泌