Exposure to the trichloroethylene metabolite S-(1,2-dichlorovinyl)-L-cysteine under hypoxic conditions (%O(2) =2 %) alters differential gene expression and mitigates decreased invasion capacity, compared to normoxic conditions (%O(2) ≥ 21 %) in HTR-8/SVneo cells

在低氧条件（%O₂ = 2%）下，三氯乙烯代谢物S-(1,2-二氯乙烯基)-L-半胱氨酸的暴露会改变HTR-8/SVneo细胞的差异基因表达，并减轻侵袭能力的下降，与常氧条件（%O₂ ≥ 21%）相比。