日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Expression of human A53T alpha-synuclein without endogenous rat alpha-synuclein fails to elicit Parkinson's disease-related phenotypes in a novel humanized rat model.

在新型人源化大鼠模型中,表达人类 A53T α-突触核蛋白而没有内源性大鼠 α-突触核蛋白,无法引起帕金森病相关表型

Polinski Nicole K, Puoliväli Jukka, Rauhala Leena, Stenius Taina-Kaisa, Bragge Timo, Parkkari Teija, Penttinen Anna-Maija, Chen Yi, Mabrouk Omar S, Glajch Kelly E, Hirst Warren D, Perkinton Michael, Martinez Terina N, Frasier Mark A

A strategy for the generation, characterization and distribution of animal models by The Michael J. Fox Foundation for Parkinson's Research

迈克尔·J·福克斯帕金森研究基金会动物模型生成、表征和分发策略

Baptista, Marco A S; Dave, Kuldip D; Sheth, Niketa P; De Silva, Shehan N; Carlson, Kirsten M; Aziz, Yasmin N; Fiske, Brian K; Sherer, Todd B; Frasier, Mark A

Loss of leucine-rich repeat kinase 2 (LRRK2) in rats leads to progressive abnormal phenotypes in peripheral organs

大鼠体内富含亮氨酸重复激酶2(LRRK2)的缺失会导致外周器官出现进行性异常表型。

Baptista, Marco A S; Dave, Kuldip D; Frasier, Mark A; Sherer, Todd B; Greeley, Melanie; Beck, Melissa J; Varsho, Julie S; Parker, George A; Moore, Cindy; Churchill, Madeline J; Meshul, Charles K; Fiske, Brian K