日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

MAX inactivation deregulates the MYC network and induces neuroendocrine neoplasia in multiple tissues.

MAX失活会扰乱MYC网络,并在多种组织中诱发神经内分泌肿瘤

Freie Brian, Ibrahim Ali H, Carroll Patrick A, Bronson Roderick T, Augert Arnaud, MacPherson David, Eisenman Robert N

Corrigendum: A germline point mutation in the MYC-FBW7 phosphodegron initiates hematopoietic malignancies

更正:MYC-FBW7磷酸化降解子中的种系点突变引发造血系统恶性肿瘤

Freie, Brian; Carroll, Patrick A; Varnum-Finney, Barbara J; Ramsey, Erin L; Ramani, Vijay; Bernstein, Irwin; Eisenman, Robert N

MondoA mediates transcriptional coordination between the MYC network and the integrated stress response in pancreatic ductal adenocarcinoma

MondoA介导MYC网络与胰腺导管腺癌整合应激反应之间的转录协调

Ramsey, Erin L; Dobersch, Stephanie; Freie, Brian; Hong, Nan Hyung; Wu, Xiaoying; Kugel, Sita; Eisenman, Robert N; Carroll, Patrick A

Max deletion destabilizes MYC protein and abrogates Eµ-Myc lymphomagenesis

Max基因缺失会破坏MYC蛋白的稳定性并抑制Eµ-Myc淋巴瘤的发生。

Mathsyaraja, Haritha; Freie, Brian; Cheng, Pei-Feng; Babaeva, Ekaterina; Catchpole, Jonathen T; Janssens, Derek; Henikoff, Steven; Eisenman, Robert N

The MYC transcription factor network: balancing metabolism, proliferation and oncogenesis

MYC转录因子网络:平衡代谢、增殖和肿瘤发生

Carroll, Patrick A; Freie, Brian W; Mathsyaraja, Haritha; Eisenman, Robert N

Ex vivo culture of Fancc-/- stem/progenitor cells predisposes cells to undergo apoptosis, and surviving stem/progenitor cells display cytogenetic abnormalities and an increased risk of malignancy

体外培养Fancc-/-干/祖细胞会使细胞易于发生凋亡,而存活的干/祖细胞则表现出细胞遗传学异常和恶性肿瘤风险增加。

Li, Xiaxin; Le Beau, Michelle M; Ciccone, Samantha; Yang, Feng-Chun; Freie, Brian; Chen, Shi; Yuan, Jin; Hong, Ping; Orazi, Attilio; Haneline, Laura S; Clapp, D Wade