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Rumian Nicole L, Barker C Madison, Larsen Matthew E, Tullis Jonathan E, Freund Ronald K, Taslimi Amir, Coultrap Steven J, Tucker Chandra L, Dell'Acqua Mark L, Bayer K Ulrich

其它

发表日期：2024

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Amyloid-Î² Causes NMDA Receptor Dysfunction and Dendritic Spine Loss through mGluR1 and AKAP150-Anchored Calcineurin Signaling.

淀粉样蛋白β通过mGluR1和AKAP150锚定钙调磷酸酶信号传导导致NMDA受体功能障碍和树突棘丢失

期刊:Journal of Neuroscience

影响因子:4

doi:10.1523/JNEUROSCI.0675-24.2024

Prikhodko Olga, Freund Ronald K, Sullivan Emily, Kennedy Matthew J, Dell'Acqua Mark L

免疫/内分泌

发表日期：2024

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LTP induction by structural rather than enzymatic functions of CaMKII

CaMKII 的结构功能而非酶功能诱导 LTP

期刊:Nature

影响因子:48.5

doi:10.1038/s41586-023-06465-y

Tullis, Jonathan E; Larsen, Matthew E; Rumian, Nicole L; Freund, Ronald K; Boxer, Emma E; Brown, Carolyn Nicole; Coultrap, Steven J; Schulman, Howard; Aoto, Jason; Dell'Acqua, Mark L; Bayer, K Ulrich

发表日期：2023

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Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice

CaMKII亚硝基化水平降低会导致小鼠出现与衰老相关的记忆力和突触可塑性障碍。

期刊:Science Signaling

影响因子:6.6

doi:10.1126/scisignal.ade5892

Rumian, Nicole L; Freund, Ronald K; Dell'Acqua, Mark L; Coultrap, Steven J; Bayer, K Ulrich

Inhibition of the Motor Protein Eg5/Kinesin-5 in Amyloid β-Mediated Impairment of Hippocampal Long-Term Potentiation and Dendritic Spine Loss

淀粉样蛋白β介导的海马长时程增强和树突棘丢失中运动蛋白Eg5/Kinesin-5的抑制

期刊:Molecular Pharmacology

影响因子:3

doi:10.1124/mol.115.103085

Freund, Ronald K; Gibson, Emily S; Potter, Huntington; Dell'Acqua, Mark L

Hippo

发表日期：2016

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Genetic knockout of the α7 nicotinic acetylcholine receptor gene alters hippocampal long-term potentiation in a background strain-dependent manner

α7尼古丁乙酰胆碱受体基因的基因敲除会以背景品系依赖的方式改变海马长时程增强作用。

期刊:Neuroscience Letters

影响因子:2

doi:10.1016/j.neulet.2016.05.043

Freund, Ronald K; Graw, Sharon; Choo, Kevin S; Stevens, Karen E; Leonard, Sherry; Dell'Acqua, Mark L

信号转导

发表日期：2016

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Blocking leukotriene synthesis attenuates the pathophysiology of traumatic brain injury and associated cognitive deficits

抑制白三烯合成可减轻创伤性脑损伤的病理生理反应及相关认知缺陷。

期刊:Experimental Neurology

影响因子:4.2

doi:10.1016/j.expneurol.2014.03.008

Corser-Jensen, Chelsea E; Goodell, Dayton J; Freund, Ronald K; Serbedzija, Predrag; Murphy, Robert C; Farias, Santiago E; Dell'Acqua, Mark L; Frey, Lauren C; Serkova, Natalie; Heidenreich, Kim A

神经科学

发表日期：2014

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LTP expression mediated by autonomous activity of GluN2B-bound CaMKII.

LTP表达由GluN2B结合的CaMKII的自主活性介导

Amyloid-Î² Causes NMDA Receptor Dysfunction and Dendritic Spine Loss through mGluR1 and AKAP150-Anchored Calcineurin Signaling.

淀粉样蛋白β通过mGluR1和AKAP150锚定钙调磷酸酶信号传导导致NMDA受体功能障碍和树突棘丢失

LTP induction by structural rather than enzymatic functions of CaMKII

CaMKII 的结构功能而非酶功能诱导 LTP

Decreased nitrosylation of CaMKII causes aging-associated impairments in memory and synaptic plasticity in mice

CaMKII亚硝基化水平降低会导致小鼠出现与衰老相关的记忆力和突触可塑性障碍。

Inhibition of the Motor Protein Eg5/Kinesin-5 in Amyloid β-Mediated Impairment of Hippocampal Long-Term Potentiation and Dendritic Spine Loss

淀粉样蛋白β介导的海马长时程增强和树突棘丢失中运动蛋白Eg5/Kinesin-5的抑制

Genetic knockout of the α7 nicotinic acetylcholine receptor gene alters hippocampal long-term potentiation in a background strain-dependent manner

α7尼古丁乙酰胆碱受体基因的基因敲除会以背景品系依赖的方式改变海马长时程增强作用。

Blocking leukotriene synthesis attenuates the pathophysiology of traumatic brain injury and associated cognitive deficits

抑制白三烯合成可减轻创伤性脑损伤的病理生理反应及相关认知缺陷。