Fritz Gerhard相关文献与解析，生知库 | 链接生命科学的每一步

Overcoming acquired doxorubicin resistance of ovarian carcinoma cells by verapamilâmediated promotion of DNA damageâdriven cytotoxicity.

通过维拉帕米介导的DNA损伤驱动的细胞毒性促进作用，克服卵巢癌细胞获得的阿霉素耐药性。

doi:10.3892/ijo.2026.5861

Mukinovic Elvira, Federmann Sina, Messling Larissa, Sekeres Marlena, Mann Julia, Abbey Lena, Kassack Matthias U, Fritz Gerhard

Combined inhibition of RAD51 and CHK1 causes synergistic toxicity in cisplatin resistant cancer cells by triggering replication fork collapse.

RAD51 和 CHK1 的联合抑制可通过触发复制叉崩溃，对顺铂耐药癌细胞产生协同毒性

期刊:International Journal of Cancer

影响因子:4.7

doi:10.1002/ijc.35164

Mann Julia, Niedermayer Kathrin, Krautstrunk Johannes, Abbey Lena, WiesmÃ¼ller Lisa, Piekorz Roland P, Fritz Gerhard

Evaluation of Genotoxic Effects of N-Methyl-N-Nitroso-Urea and Etoposide on the Differentiation Potential of MSCs from Umbilical Cord Blood and Bone Marrow

评价N-甲基-N-亚硝基脲和依托泊苷对脐带血和骨髓间充质干细胞分化潜能的遗传毒性作用

期刊:Cells

影响因子:5.2

doi:10.3390/cells13242134

Ouzin, Meryem; Wesselborg, Sebastian; Fritz, Gerhard; Kogler, Gesine

EWSR1-NFATC2 and FUS-NFATC2 Gene Fusion-Associated Mesenchymal Tumors: Clinicopathologic Correlation and Literature Review

EWSR1-NFATC2 和 FUS-NFATC2 基因融合相关间叶肿瘤：临床病理相关性和文献综述

期刊:Sarcoma

影响因子:

doi:10.1155/2019/9386390

Beata Bode-Lesniewska, Christine Fritz, Gerhard Ulrich Exner, Ulrich Wagner, Bruno Fuchs

肿瘤

发表日期：2019

文献求助收藏

Statins in anthracycline-induced cardiotoxicity: Rac and Rho, and the heartbreakers

他汀类药物在蒽环类药物诱发的心脏毒性中的应用：Rac 和 Rho，以及那些令人心碎的疾病

期刊:Cell Death & Disease

影响因子:9.6

doi:10.1038/cddis.2016.418

Henninger, Christian; Fritz, Gerhard

毒理研究

发表日期：2017

文献求助收藏

Rho GTPases: Novel Players in the Regulation of the DNA Damage Response?

Rho GTP酶：DNA损伤反应调控中的新角色？

期刊:Biomolecules

影响因子:4.8

doi:10.3390/biom5042417

Fritz, Gerhard; Henninger, Christian

TSG (2,3,5,4'-Tetrahydroxystilbene-2-O- β -D-glucoside) from the Chinese Herb Polygonum multiflorum Increases Life Span and Stress Resistance of Caenorhabditis elegans

来自中药何首乌的TSG（2,3,5,4'-四羟基芪-2-O-β-D-葡萄糖苷）可延长秀丽隐杆线虫的寿命并提高其抗逆性

期刊:Oxidative Medicine and Cellular Longevity

影响因子:

doi:10.1155/2015/124357

Büchter, Christian; Zhao, Liang; Havermann, Susannah; Honnen, Sebastian; Fritz, Gerhard; Proksch, Peter; Wätjen, Wim

C. elegans

发表日期：2015

文献求助收藏

P38 MAP kinase signaling is required for the conversion of CD4+CD25- T cells into iTreg

p38 MAP激酶信号通路是CD4+CD25- T细胞转化为iTreg细胞所必需的。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0003302

Huber, Samuel; Schrader, Jörg; Fritz, Gerhard; Presser, Katrin; Schmitt, Steffen; Waisman, Ari; Lüth, Stefan; Blessing, Manfred; Herkel, Johannes; Schramm, Christoph

Late activation of stress kinases (SAPK/JNK) by genotoxins requires the DNA repair proteins DNA-PKcs and CSB

基因毒素引起的应激激酶（SAPK/JNK）的后期激活需要DNA修复蛋白DNA-PKcs和CSB。

期刊:Molecular Biology of the Cell

影响因子:2.7

doi:10.1091/mbc.e05-07-0606

Fritz, Gerhard; Kaina, Bernd

Sublethal DNA damage switches off B cell effector programs in an RA-FLS-PBMC co-culture

亚致死性DNA损伤会关闭RA-FLS-PBMC共培养体系中的B细胞效应程序。

Overcoming acquired doxorubicin resistance of ovarian carcinoma cells by verapamilâmediated promotion of DNA damageâdriven cytotoxicity.

通过维拉帕米介导的DNA损伤驱动的细胞毒性促进作用，克服卵巢癌细胞获得的阿霉素耐药性。

Combined inhibition of RAD51 and CHK1 causes synergistic toxicity in cisplatin resistant cancer cells by triggering replication fork collapse.

RAD51 和 CHK1 的联合抑制可通过触发复制叉崩溃，对顺铂耐药癌细胞产生协同毒性

Evaluation of Genotoxic Effects of N-Methyl-N-Nitroso-Urea and Etoposide on the Differentiation Potential of MSCs from Umbilical Cord Blood and Bone Marrow

评价N-甲基-N-亚硝基脲和依托泊苷对脐带血和骨髓间充质干细胞分化潜能的遗传毒性作用

EWSR1-NFATC2 and FUS-NFATC2 Gene Fusion-Associated Mesenchymal Tumors: Clinicopathologic Correlation and Literature Review

EWSR1-NFATC2 和 FUS-NFATC2 基因融合相关间叶肿瘤：临床病理相关性和文献综述

Statins in anthracycline-induced cardiotoxicity: Rac and Rho, and the heartbreakers

他汀类药物在蒽环类药物诱发的心脏毒性中的应用：Rac 和 Rho，以及那些令人心碎的疾病

Rho GTPases: Novel Players in the Regulation of the DNA Damage Response?

Rho GTP酶：DNA损伤反应调控中的新角色？

TSG (2,3,5,4'-Tetrahydroxystilbene-2-O- β -D-glucoside) from the Chinese Herb Polygonum multiflorum Increases Life Span and Stress Resistance of Caenorhabditis elegans

来自中药何首乌的TSG（2,3,5,4'-四羟基芪-2-O-β-D-葡萄糖苷）可延长秀丽隐杆线虫的寿命并提高其抗逆性

P38 MAP kinase signaling is required for the conversion of CD4+CD25- T cells into iTreg

p38 MAP激酶信号通路是CD4+CD25- T细胞转化为iTreg细胞所必需的。

Late activation of stress kinases (SAPK/JNK) by genotoxins requires the DNA repair proteins DNA-PKcs and CSB

基因毒素引起的应激激酶（SAPK/JNK）的后期激活需要DNA修复蛋白DNA-PKcs和CSB。

Sublethal DNA damage switches off B cell effector programs in an RA-FLS-PBMC co-culture

亚致死性DNA损伤会关闭RA-FLS-PBMC共培养体系中的B细胞效应程序。

Overcoming acquired doxorubicin resistance of ovarian carcinoma cells by verapamilâmediated promotion of DNA damageâdriven cytotoxicity.

通过维拉帕米介导的DNA损伤驱动的细胞毒性促进作用，克服卵巢癌细胞获得的阿霉素耐药性。

Combined inhibition of RAD51 and CHK1 causes synergistic toxicity in cisplatin resistant cancer cells by triggering replication fork collapse.

RAD51 和 CHK1 的联合抑制可通过触发复制叉崩溃，对顺铂耐药癌细胞产生协同毒性

Evaluation of Genotoxic Effects of N-Methyl-N-Nitroso-Urea and Etoposide on the Differentiation Potential of MSCs from Umbilical Cord Blood and Bone Marrow

评价N-甲基-N-亚硝基脲和依托泊苷对脐带血和骨髓间充质干细胞分化潜能的遗传毒性作用

EWSR1-NFATC2 and FUS-NFATC2 Gene Fusion-Associated Mesenchymal Tumors: Clinicopathologic Correlation and Literature Review

EWSR1-NFATC2 和 FUS-NFATC2 基因融合相关间叶肿瘤：临床病理相关性和文献综述

Statins in anthracycline-induced cardiotoxicity: Rac and Rho, and the heartbreakers

他汀类药物在蒽环类药物诱发的心脏毒性中的应用：Rac 和 Rho，以及那些令人心碎的疾病

Rho GTPases: Novel Players in the Regulation of the DNA Damage Response?

Rho GTP酶：DNA损伤反应调控中的新角色？

TSG (2,3,5,4'-Tetrahydroxystilbene-2-O- β -D-glucoside) from the Chinese Herb Polygonum multiflorum Increases Life Span and Stress Resistance of Caenorhabditis elegans

来自中药何首乌的TSG（2,3,5,4'-四羟基芪-2-O-β-D-葡萄糖苷）可延长秀丽隐杆线虫的寿命并提高其抗逆性

P38 MAP kinase signaling is required for the conversion of CD4+CD25- T cells into iTreg

p38 MAP激酶信号通路是CD4+CD25- T细胞转化为iTreg细胞所必需的。

Late activation of stress kinases (SAPK/JNK) by genotoxins requires the DNA repair proteins DNA-PKcs and CSB

基因毒素引起的应激激酶（SAPK/JNK）的后期激活需要DNA修复蛋白DNA-PKcs和CSB。

Overcoming acquired doxorubicin resistance of ovarian carcinoma cells by verapamilâmediated promotion of DNA damageâdriven cytotoxicity.