Fu Ssu-Ju相关文献与解析，生知库 | 链接生命科学的每一步

Li Jeng-Lin, Huang Shu-Yi, Huang Po-Yu, Fu Ssu-Ju, Tsao Yu-Jung, Chang Wenying, Hong Cheng-Li, Hung Yu-Chien, Liu Pei-Han, Lai Liang-Chuan, Lin Chin-Hsien, Chiang Wei-Chung, Chan Chih-Chiang

帕金森

表观遗传

NK1

Restoration of Shal/K(V)4 proteostasis and motor function in a Drosophila model of spinocerebellar ataxia type 19/22.

在果蝇脊髓小脑性共济失调 19/22 型模型中恢复 Shal/K(V)4 蛋白稳态和运动功能

期刊:Cellular and Molecular Life Sciences

影响因子:6.2

doi:10.1007/s00018-025-05711-y

Hsiao Cheng-Tsung, Fu Ssu-Ju, Cheng Kai-Min, Lo Hsiang, Tang Chih-Yung, Chan Chih-Chiang, Jeng Chung-Jiuan

A fly model of SCA36 reveals combinatorial neurotoxicity of hexanucleotide and dipeptide repeats.

SCA36 果蝇模型揭示了六核苷酸和二肽重复序列的组合神经毒性。

期刊:PLoS Genetics

影响因子:3.7

doi:10.1371/journal.pgen.1011954

Hsiao Cheng-Tsung, Fu Ssu-Ju, Guo Ting-Ni, Lin Chia-Chi, Tsao Yu-Jung, Chang Wenying, Liao Yi-Chu, Hashimoto Masayuki, Huang Shu-Yi, Lee Yi-Chung, Yu Chien-Hung, Chan Chih-Chiang

神经科学

发表日期：2025

文献求助收藏

Regulation of testosterone synthesis in Leydig cells by ClC-2 chloride channel.

ClC-2氯离子通道对睾丸间质细胞中睾酮合成的调节

期刊:Reproduction

影响因子:3.7

doi:10.1530/REP-24-0432

Fu Ssu-Ju, Syu Min-Shan, Tang Chih-Yu, Huang Ching-Yuan, Jeng Chung-Jiuan, Tang Chih-Yung, Hu Meng-Chun

细胞生物学

发表日期：2025

文献求助收藏

Novel KCND3 Variant Underlying Nonprogressive Congenital Ataxia or SCA19/22 Disrupt K(V)4.3 Protein Expression and K+ Currents with Variable Effects on Channel Properties

一种导致非进行性先天性共济失调或SCA19/22的新型KCND3变异体，会破坏K(V)4.3蛋白的表达和K+电流，并对通道特性产生不同的影响。

期刊:International Journal of Molecular Sciences

影响因子:4.9

doi:10.3390/ijms22094986

Zanni, Ginevra; Hsiao, Cheng-Tsung; Fu, Ssu-Ju; Tang, Chih-Yung; Capuano, Alessandro; Bosco, Luca; Graziola, Federica; Bellacchio, Emanuele; Servidei, Serenella; Primiano, Guido; Soong, Bing-Wen; Jeng, Chung-Jiuan

发表日期：2021

文献求助收藏

Defective Gating and Proteostasis of Human ClC-1 Chloride Channel: Molecular Pathophysiology of Myotonia Congenita

人类ClC-1氯离子通道的门控和蛋白稳态缺陷：先天性肌强直的分子病理生理学

期刊:Frontiers in Neurology

影响因子:2.8

doi:10.3389/fneur.2020.00076

Jeng, Chung-Jiuan; Fu, Ssu-Ju; You, Chia-Ying; Peng, Yi-Jheng; Hsiao, Cheng-Tsung; Chen, Tsung-Yu; Tang, Chih-Yung

Human

发表日期：2020

文献求助收藏

The episodic ataxia type 1 mutation I262T alters voltage-dependent gating and disrupts protein biosynthesis of human Kv1.1 potassium channels

发作性共济失调1型突变I262T会改变电压依赖性门控并破坏人Kv1.1钾通道的蛋白质生物合成。

期刊:Scientific Reports

影响因子:3.9

doi:10.1038/srep19378

Chen, Szu-Han; Fu, Ssu-Ju; Huang, Jing-Jia; Tang, Chih-Yung

发表日期：2016

文献求助收藏

The Eag domain regulates the voltage-dependent inactivation of rat Eag1 K+ channels

Eag结构域调控大鼠Eag1 K+通道的电压依赖性失活。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0110423

Lin, Ting-Feng; Jow, Guey-Mei; Fang, Hsin-Yu; Fu, Ssu-Ju; Wu, Hao-Han; Chiu, Mei-Miao; Jeng, Chung-Jiuan

UQCRC1 deficiency impairs mitophagy via PINK1-dependent mechanisms in Parkinson's disease.

UQCRC1 缺陷通过 PINK1 依赖性机制损害帕金森病中的线粒体自噬。

Restoration of Shal/K(V)4 proteostasis and motor function in a Drosophila model of spinocerebellar ataxia type 19/22.

在果蝇脊髓小脑性共济失调 19/22 型模型中恢复 Shal/K(V)4 蛋白稳态和运动功能

A fly model of SCA36 reveals combinatorial neurotoxicity of hexanucleotide and dipeptide repeats.

SCA36 果蝇模型揭示了六核苷酸和二肽重复序列的组合神经毒性。

Regulation of testosterone synthesis in Leydig cells by ClC-2 chloride channel.

ClC-2氯离子通道对睾丸间质细胞中睾酮合成的调节

Novel KCND3 Variant Underlying Nonprogressive Congenital Ataxia or SCA19/22 Disrupt K(V)4.3 Protein Expression and K+ Currents with Variable Effects on Channel Properties

一种导致非进行性先天性共济失调或SCA19/22的新型KCND3变异体，会破坏K(V)4.3蛋白的表达和K+电流，并对通道特性产生不同的影响。

Defective Gating and Proteostasis of Human ClC-1 Chloride Channel: Molecular Pathophysiology of Myotonia Congenita

人类ClC-1氯离子通道的门控和蛋白稳态缺陷：先天性肌强直的分子病理生理学

The episodic ataxia type 1 mutation I262T alters voltage-dependent gating and disrupts protein biosynthesis of human Kv1.1 potassium channels

发作性共济失调1型突变I262T会改变电压依赖性门控并破坏人Kv1.1钾通道的蛋白质生物合成。

The Eag domain regulates the voltage-dependent inactivation of rat Eag1 K+ channels

Eag结构域调控大鼠Eag1 K+通道的电压依赖性失活。