日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

HSF1 and HSF3 cooperatively regulate the heat shock response in lizards

HSF1和HSF3协同调节蜥蜴的热休克反应

Takii, Ryosuke; Fujimoto, Mitsuaki; Matsuura, Yuki; Wu, Fangxu; Oshibe, Namiko; Takaki, Eiichi; Katiyar, Arpit; Akashi, Hiroshi; Makino, Takashi; Kawata, Masakado; Nakai, Akira

Heat shock factor 1 contributes to ischemia-induced angiogenesis by regulating the mobilization and recruitment of bone marrow stem/progenitor cells

热休克因子1通过调节骨髓干/祖细胞的动员和募集,促进缺血诱导的血管生成。

Kubo, Masayuki; Li, Tao-Sheng; Kurazumi, Hiroshi; Takemoto, Yoshihiro; Ohshima, Mako; Yamamoto, Yumi; Nishimoto, Arata; Mikamo, Akihito; Fujimoto, Mitsuaki; Nakai, Akira; Hamano, Kimikazu

Impaired hippocampal spinogenesis and neurogenesis and altered affective behavior in mice lacking heat shock factor 1

缺乏热休克因子1的小鼠海马棘突生成和神经发生受损,以及情感行为改变

Uchida, Shusaku; Hara, Kumiko; Kobayashi, Ayumi; Fujimoto, Mitsuaki; Otsuki, Koji; Yamagata, Hirotaka; Hobara, Teruyuki; Abe, Naoko; Higuchi, Fumihiro; Shibata, Tomohiko; Hasegawa, Shunsuke; Kida, Satoshi; Nakai, Akira; Watanabe, Yoshifumi

Transcription factor cooperativity with heat shock factor 1

转录因子与热休克因子1的协同作用

Hayashida, Naoki; Fujimoto, Mitsuaki; Nakai, Akira

Heat shock factor 2 is required for maintaining proteostasis against febrile-range thermal stress and polyglutamine aggregation

热休克因子2对于维持蛋白质稳态,抵抗发热范围内的热应激和多聚谷氨酰胺聚集至关重要。

Shinkawa, Toyohide; Tan, Ke; Fujimoto, Mitsuaki; Hayashida, Naoki; Yamamoto, Kaoru; Takaki, Eiichi; Takii, Ryosuke; Prakasam, Ramachandran; Inouye, Sachiye; Mezger, Valerie; Nakai, Akira

Heat shock factor 1 ameliorates proteotoxicity in cooperation with the transcription factor NFAT

热休克因子1与转录因子NFAT协同作用,减轻蛋白毒性。

Hayashida, Naoki; Fujimoto, Mitsuaki; Tan, Ke; Prakasam, Ramachandran; Shinkawa, Toyohide; Li, Liangping; Ichikawa, Hitoshi; Takii, Ryosuke; Nakai, Akira

A novel mouse HSF3 has the potential to activate nonclassical heat-shock genes during heat shock.

一种新型小鼠 HSF3 有可能在热休克期间激活非经典热休克基因

Fujimoto Mitsuaki, Hayashida Naoki, Katoh Takuma, Oshima Kouji, Shinkawa Toyohide, Prakasam Ramachandran, Tan Ke, Inouye Sachiye, Takii Ryosuke, Nakai Akira

Analysis of HSF4 binding regions reveals its necessity for gene regulation during development and heat shock response in mouse lenses

对 HSF4 结合区域的分析揭示了其在小鼠晶状体发育和热休克反应过程中基因调控的必要性

Fujimoto, Mitsuaki; Oshima, Koji; Shinkawa, Toyohide; Wang, Bei Bei; Inouye, Sachiye; Hayashida, Naoki; Takii, Ryosuke; Nakai, Akira

A novel HSF1-mediated death pathway that is suppressed by heat shock proteins

一种新型的HSF1介导的细胞死亡通路,可被热休克蛋白抑制

Hayashida, Naoki; Inouye, Sachiye; Fujimoto, Mitsuaki; Tanaka, Yasunori; Izu, Hanae; Takaki, Eiichi; Ichikawa, Hitoshi; Rho, Jaerang; Nakai, Akira

HSF4 is required for normal cell growth and differentiation during mouse lens development

HSF4是小鼠晶状体发育过程中正常细胞生长和分化所必需的

Fujimoto, Mitsuaki; Izu, Hanae; Seki, Keisuke; Fukuda, Ken; Nishida, Teruo; Yamada, Shu-Ichi; Kato, Kanefusa; Yonemura, Shigenobu; Inouye, Sachiye; Nakai, Akira