日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Defective DNA Damage Response is a Targetable Therapeutic Vulnerability in ESR1 Mutant Breast Cancer.

DNA损伤反应缺陷是ESR1突变型乳腺癌的一个可靶向治疗弱点。

Herzog Sarah K, Stevens Jessica H, Gu Guowei, Grimm Sandra L, Cardoza Kloma, Hawkins Autumn G M, Lin Hangqing, Ramos Daniela, Beyer Amanda R, Edwards David G, Dustin Derek, Yang Harry J, Liang Nicole, Khan Ashfia F, Bawa-Khalfe Tasneem, McGrail Daniel J, Lin Shiaw-Yih, Coarfa Cristian, Fuqua Suzanne A W

Adipocyte/Tumor cell crosstalk via IGF-1/TXNIP axis promotes malignancy and endocrine resistance in breast cancer.

脂肪细胞/肿瘤细胞通过 IGF-1/TXNIP 轴进行相互作用,促进乳腺癌的恶性转化和内分泌耐药性

Caruso Amanda, Accattatis Felice Maria, Giordano Cinzia, Gelsomino Luca, Del Console Piercarlo, Fiorita Maria Francesca, Gyorffy Balazs, Bianchi Laura, Carleo Alfonso, De Salvo Rossana, Simões Bruno M, Clarke Robert B, Memeo Lorenzo, Colarossi Cristina, Aiello Eleonora, Conforti Francesco, Fuqua Suzanne A W, Todaro Matilde, Stassi Giorgio, Morelli Catia, Sisci Diego, Bonofiglio Daniela, Catalano Stefania, Andò Sebastiano, Barone Ines

Discovery of a novel small molecule degrader of wild type and mutant estrogen receptors using DNA encoded libraries

利用DNA编码文库发现一种新型小分子降解剂,可降解野生型和突变型雌激素受体。

Devakrishnan, Anil Kumar; Madasu, Chandrashekhar; Wang, Yong; Modukuri, Ramkumar; Bohren, Kurt M; MacKenzie, Kevin R; Young, Damian W; Fuqua, Suzanne A W; Matzuk, Martin M; Palaniappan, Murugesan

Correction: RON signalling promotes therapeutic resistance in ESR1 mutant breast cancer

更正:RON信号通路促进ESR1突变型乳腺癌的治疗耐药性

Dustin, Derek; Gu, Guowei; Beyer, Amanda R; Herzog, Sarah K; Edwards, David G; Lin, Hangqing; Gonzalez, Thomas L; Grimm, Sandra L; Coarfa, Cristian; Chan, Doug W; Kim, Beom-Jun; De La O, Jean-Paul; Ellis, Matthew J; Liu, Dan; Li, Shunqiang; Welm, Alana L; Fuqua, Suzanne A W

Correction to: Hormonal modulation of ESR1 mutant metastasis

更正:激素对 ESR1 突变转移的调节

Gu, Guowei; Tian, Lin; Herzog, Sarah K; Rechoum, Yassine; Gelsomino, Luca; Gao, Meng; Du, Lili; Kim, Jin-Ah; Dustin, Derek; Lo, Hin Ching; Beyer, Amanda R; Edwards, David G; Gonzalez, Thomas; Tsimelzon, Anna; Huang, Helen J; Fernandez, Natalie M; Grimm, Sandra L; Hilsenbeck, Susan G; Liu, Dan; Xu, Jun; Alaniz, Alyssa; Li, Shunqiang; Mills, Gordon B; Janku, Filip; Kittler, Ralf; Zhang, Xiang H-F; Coarfa, Cristian; Foulds, Charles E; Symmans, W Fraser; Andò, Sebastiano; Fuqua, Suzanne A W

ESR1 mutations and therapeutic resistance in metastatic breast cancer: progress and remaining challenges

ESR1突变与转移性乳腺癌的治疗耐药性:进展与挑战

Herzog, Sarah K; Fuqua, Suzanne A W

Hormonal modulation of ESR1 mutant metastasis

激素对ESR1突变转移的调节

Gu, Guowei; Tian, Lin; Herzog, Sarah K; Rechoum, Yassine; Gelsomino, Luca; Gao, Meng; Du, Lili; Kim, Jin-Ah; Dustin, Derek; Lo, Hin Ching; Beyer, Amanda R; Edwards, David G; Gonzalez, Thomas; Tsimelzon, Anna; Huang, Helen J; Fernandez, Natalie M; Grimm, Sandra L; Hilsenbeck, Susan G; Liu, Dan; Xu, Jun; Alaniz, Alyssa; Li, Shunqiang; Mills, Gordon B; Janku, Filip; Kittler, Ralf; Zhang, Xiang H-F; Coarfa, Cristian; Foulds, Charles E; Symmans, W Fraser; Andò, Sebastiano; Fuqua, Suzanne A W

Estrogen-induced transcription at individual alleles is independent of receptor level and active conformation but can be modulated by coactivators activity

雌激素诱导的单个等位基因转录与受体水平和活性构象无关,但可被辅激活因子活性调节。

Stossi, Fabio; Dandekar, Radhika D; Mancini, Maureen G; Gu, Guowei; Fuqua, Suzanne A W; Nardone, Agostina; De Angelis, Carmine; Fu, Xiaoyong; Schiff, Rachel; Bedford, Mark T; Xu, Wei; Johansson, Hans E; Stephan, Clifford C; Mancini, Michael A

Erratum to: ESR1 mutations affect anti-proliferative responses to tamoxifen through enhanced cross-talk with IGF signaling

更正:ESR1 突变通过增强与 IGF 信号的串扰影响他莫昔芬的抗增殖反应

Gelsomino, Luca; Gu, Guowei; Rechoum, Yassine; Beyer, Amanda R; Pejerrey, Sasha M; Tsimelzon, Anna; Wang, Tao; Huffman, Kenneth; Ludlow, Andrew; Andò, Sebastiano; Fuqua, Suzanne A W

ESR1 Mutations in Cell-Free DNA of Breast Cancer: Predictive "Tip of the Iceberg"

乳腺癌细胞游离DNA中的ESR1突变:预测性的“冰山一角”

Fuqua, Suzanne A W; Rechoum, Yassine; Gu, Guowei