日期:
2020 年 — 2026 年
2020
2021
2022
2023
2024
2025
2026
影响因子:

Suppressor Mutations for Presenilin 1 Familial Alzheimer Disease Mutants Modulate γ-Secretase Activities.

抑制早老素 1 家族性阿尔茨海默病突变体的突变调节 α-分泌酶活性

Futai Eugene, Osawa Satoko, Cai Tetsuo, Fujisawa Tomoya, Ishiura Shoichi, Tomita Taisuke

Sec24p and Sec16p cooperate to regulate the GTP cycle of the COPII coat

Sec24p 和 Sec16p 协同调节 COPII 外壳的 GTP 循环。

Kung, Leslie F; Pagant, Silvere; Futai, Eugene; D'Arcangelo, Jennifer G; Buchanan, Roy; Dittmar, John C; Reid, Robert J D; Rothstein, Rodney; Hamamoto, Susan; Snapp, Erik L; Schekman, Randy; Miller, Elizabeth A

Comparison of presenilin 1 and presenilin 2 γ-secretase activities using a yeast reconstitution system

利用酵母重组系统比较早老素1和早老素2的γ-分泌酶活性

Yonemura, Yoji; Futai, Eugene; Yagishita, Sosuke; Suo, Satoshi; Tomita, Taisuke; Iwatsubo, Takeshi; Ishiura, Shoichi

beta-Secretase inhibitor potency is decreased by aberrant beta-cleavage location of the "Swedish mutant" amyloid precursor protein.

β-分泌酶抑制剂的效力因“瑞典突变”淀粉样前体蛋白的异常β-裂解位置而降低

Yamakawa Hidekuni, Yagishita Sosuke, Futai Eugene, Ishiura Shoichi

Nicastrin is dispensable for gamma-secretase protease activity in the presence of specific presenilin mutations.

在存在特定早老素突变的情况下,Nicastrin 对 γ-分泌酶蛋白酶活性并非必需

Futai Eugene, Yagishita Sosuke, Ishiura Shoichi