Gaestel Matthias相关文献与解析，生知库 | 链接生命科学的每一步

Herr Sarah M, Stalkopf Diana, Padaszus Sofie, Herbst Lukas A, DÃ¶rrie Anneke, Niedenthal Rainer, Ronkina Natalia, Yakovleva Tatiana, Kotlyarov Alexey, Gaestel Matthias

细胞生物学

巨噬细胞

P53

Targeting the TLK1-MK5 Axis Suppresses Prostate Cancer Metastasis.

靶向 TLK1-MK5 轴可抑制前列腺癌转移

期刊:Cancers

影响因子:4.4

doi:10.3390/cancers17071187

Olatunde Damilola, Franco Omar Coronel, Gaestel Matthias, De Benedetti Arrigo

MK2 deficiency decreases mortality in male mice during the inflammatory phase after myocardial infarction.

MK2 缺乏可降低雄性小鼠在心肌梗死后炎症期的死亡率。

期刊:Physiological Reports

影响因子:1.9

doi:10.14814/phy2.70558

TrÃ©panier JoÃ«lle, Nawaito Sherin A, Sahadevan Pramod, Sahmi Fatiha, Duquette Natacha, GÃ©linas Danielle, Gillis Marc-Antoine, Shi Yanfen, Torok Cynthia, Clavet-Lanthier Marie-Ãlaine, Gaestel Matthias, Sirois Martin G, Tardif Jean-Claude, Allen Bruce G

The enigma of small heat shock protein phosphorylation

小热休克蛋白磷酸化的谜团

期刊:Frontiers in Pharmacology

影响因子:4.8

doi:10.3389/fphar.2024.1486245

Enforced Dimerization of CD45 by the Adenovirus E3/49K Protein Inhibits T Cell Receptor Signaling

腺病毒E3/49K蛋白强制CD45二聚化抑制T细胞受体信号传导

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/jvi.01898-22

Windheim, Mark; Reubold, Thomas F; Aichane, Khadija; Gaestel, Matthias; Burgert, Hans-Gerhard

Editorial: Emerging Functions of Septins-Volume II

社论：Septin蛋白的新兴功能——第二卷

期刊:Frontiers in Cell and Developmental Biology

影响因子:4.3

doi:10.3389/fcell.2022.949824

Menon, Manoj B; Gaestel, Matthias

发表日期：2022

文献求助收藏

IL-3 is essential for ICOS-L stabilization on mast cells, and sustains the IL-33-induced RORÎ³t(+) T(reg) generation via enhanced IL-6 induction.

IL-3 是肥大细胞上 ICOS-L 稳定所必需的，并且通过增强 IL-6 诱导来维持 IL-33 诱导的 RORÎ³t(+) T(reg) 生成

期刊:Immunology

影响因子:5

doi:10.1111/imm.13305

Drube Sebastian, MÃ¼ller Sylvia, Weber Franziska, Wegner Philine, BÃ¶ttcher-Loschinski Romy, Gaestel Matthias, Hutloff Andreas, Kamradt Thomas, Andreas Nico

To die or not to die: Regulatory feedback phosphorylation circuits determine receptor-interacting protein kinase-1 (RIPK1) function

生死攸关：调控反馈磷酸化回路决定受体相互作用蛋白激酶-1 (RIPK1) 的功能

期刊:Molecular and Cellular Oncology

影响因子:1.9

doi:10.1080/23723556.2017.1396389

Menon, Manoj B; Gropengießer, Julia; Ruckdeschel, Klaus; Gaestel, Matthias

Editorial: Emerging Functions of Septins

社论：隔蛋白的新兴功能

期刊:Frontiers in Cell and Developmental Biology

影响因子:4.3

doi:10.3389/fcell.2017.00073

Menon, Manoj B; Gaestel, Matthias

发表日期：2017

文献求助收藏

Mitogen-Activated Protein Kinase 2 Signaling Shapes Macrophage Plasticity in Aggregatibacter actinomycetemcomitans-Induced Bone Loss.

丝裂原活化蛋白激酶 2 信号传导在伴放线放线杆菌诱导的骨丢失中塑造巨噬细胞可塑性

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00552-16

Herbert Bethany A, Steinkamp Heidi M, Gaestel Matthias, Kirkwood Keith L

细胞生物学

发表日期：2017

文献求助收藏

MK2/p38/p53 Suppress Basal IL-1Î² and Non-Canonical NF-ÎºB Signaling in Macrophages.

MK2/p38/p53抑制巨噬细胞中的基础IL-1β和非经典NF-β信号传导。

Targeting the TLK1-MK5 Axis Suppresses Prostate Cancer Metastasis.

靶向 TLK1-MK5 轴可抑制前列腺癌转移

MK2 deficiency decreases mortality in male mice during the inflammatory phase after myocardial infarction.

MK2 缺乏可降低雄性小鼠在心肌梗死后炎症期的死亡率。

The enigma of small heat shock protein phosphorylation

小热休克蛋白磷酸化的谜团

Enforced Dimerization of CD45 by the Adenovirus E3/49K Protein Inhibits T Cell Receptor Signaling

腺病毒E3/49K蛋白强制CD45二聚化抑制T细胞受体信号传导

Editorial: Emerging Functions of Septins-Volume II

社论：Septin蛋白的新兴功能——第二卷

IL-3 is essential for ICOS-L stabilization on mast cells, and sustains the IL-33-induced RORÎ³t(+) T(reg) generation via enhanced IL-6 induction.

IL-3 是肥大细胞上 ICOS-L 稳定所必需的，并且通过增强 IL-6 诱导来维持 IL-33 诱导的 RORÎ³t(+) T(reg) 生成

To die or not to die: Regulatory feedback phosphorylation circuits determine receptor-interacting protein kinase-1 (RIPK1) function

生死攸关：调控反馈磷酸化回路决定受体相互作用蛋白激酶-1 (RIPK1) 的功能

Editorial: Emerging Functions of Septins

社论：隔蛋白的新兴功能

Mitogen-Activated Protein Kinase 2 Signaling Shapes Macrophage Plasticity in Aggregatibacter actinomycetemcomitans-Induced Bone Loss.

丝裂原活化蛋白激酶 2 信号传导在伴放线放线杆菌诱导的骨丢失中塑造巨噬细胞可塑性