Garcin Dominique相关文献与解析，生知库 | 链接生命科学的每一步

Asgari, Samira; Schlapbach, Luregn J; Anchisi, Stéphanie; Hammer, Christian; Bartha, Istvan; Junier, Thomas; Mottet-Osman, Geneviève; Posfay-Barbe, Klara M; Longchamp, David; Stocker, Martin; Cordey, Samuel; Kaiser, Laurent; Riedel, Thomas; Kenna, Tony; Long, Deborah; Schibler, Andreas; Telenti, Amalio; Tapparel, Caroline; McLaren, Paul J; Garcin, Dominique; Fellay, Jacques

炎症/感染

FIH

毒理研究

微生物学

发表日期：2017

文献求助收藏

Mismatches in the Influenza A Virus RNA Panhandle Prevent Retinoic Acid-Inducible Gene I (RIG-I) Sensing by Impairing RNA/RIG-I Complex Formation.

甲型流感病毒 RNA 柄中的错配会损害 RNA/RIG-I 复合物的形成，从而阻止视黄酸诱导基因 I (RIG-I) 的感知

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/JVI.01671-15

Anchisi StÃ©phanie, Guerra Jessica, Mottet-Osman GeneviÃ¨ve, Garcin Dominique

MAVS ubiquitination by the E3 ligase TRIM25 and degradation by the proteasome is involved in type I interferon production after activation of the antiviral RIG-I-like receptors.

MAVS 经 E3 连接酶 TRIM25 泛素化和蛋白酶体降解后，参与了抗病毒 RIG-I 样受体激活后 I 型干扰素的产生

期刊:BMC Biology

影响因子:4.5

doi:10.1186/1741-7007-10-44

Castanier CÃ©line, Zemirli Naima, Portier Alain, Garcin Dominique, BidÃ¨re Nicolas, Vazquez AimÃ©, Arnoult Damien

Mitochondrial dynamics regulate the RIG-I-like receptor antiviral pathway

线粒体动力学调控RIG-I样受体抗病毒通路

期刊:EMBO Reports

影响因子:6.2

doi:10.1038/embor.2009.258

Castanier, Céline; Garcin, Dominique; Vazquez, Aimé; Arnoult, Damien

Unpaired 5' ppp-nucleotides, as found in arenavirus double-stranded RNA panhandles, are not recognized by RIG-I

未配对的5' ppp核苷酸（例如沙粒病毒双链RNA的柄状结构中存在的核苷酸）不能被RIG-I识别。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M109.089425

Marq, Jean-Baptiste; Kolakofsky, Daniel; Garcin, Dominique

微生物学

发表日期：2010

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Activation of the beta interferon promoter by unnatural Sendai virus infection requires RIG-I and is inhibited by viral C proteins.

非天然仙台病毒感染激活β干扰素启动子需要RIG-I，并受到病毒C蛋白的抑制

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/JVI.01300-07

StrÃ¤hle Laura, Marq Jean-Baptiste, Brini Albert, Hausmann StÃ©phane, Kolakofsky Daniel, Garcin Dominique

A short peptide at the amino terminus of the Sendai virus C protein acts as an independent element that induces STAT1 instability.

仙台病毒 C 蛋白氨基末端的短肽作为独立元件，可诱导 STAT1 不稳定

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/JVI.78.16.8799-8811.2004

免疫/内分泌

发表日期：2004

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The amino-terminal extensions of the longer Sendai virus C proteins modulate pY701-Stat1 and bulk Stat1 levels independently of interferon signaling.

较长的仙台病毒 C 蛋白的氨基末端延伸部分可以独立于干扰素信号传导来调节 pY701-Stat1 和整体 Stat1 的水平

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/jvi.77.4.2321-2329.2003

Garcin Dominique, Marq Jean-Baptiste, Goodbourn Stephen, Kolakofsky Daniel

免疫/内分泌

发表日期：2003

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Sendai virus trailer RNA binds TIAR, a cellular protein involved in virus-induced apoptosis

仙台病毒尾部RNA与TIAR结合，TIAR是一种参与病毒诱导细胞凋亡的细胞蛋白

期刊:EMBO Journal

影响因子:8.3

doi:10.1093/emboj/cdf513

Iseni, Frédéric; Garcin, Dominique; Nishio, Machiko; Kedersha, Nancy; Anderson, Paul; Kolakofsky, Daniel

Complete genome sequence of Tacaribe virus

塔卡里贝病毒的完整基因组序列

Severe viral respiratory infections in children with IFIH1 loss-of-function mutations

患有 IFIH1 功能丧失突变的儿童发生严重病毒性呼吸道感染

Mismatches in the Influenza A Virus RNA Panhandle Prevent Retinoic Acid-Inducible Gene I (RIG-I) Sensing by Impairing RNA/RIG-I Complex Formation.

甲型流感病毒 RNA 柄中的错配会损害 RNA/RIG-I 复合物的形成，从而阻止视黄酸诱导基因 I (RIG-I) 的感知

MAVS ubiquitination by the E3 ligase TRIM25 and degradation by the proteasome is involved in type I interferon production after activation of the antiviral RIG-I-like receptors.

MAVS 经 E3 连接酶 TRIM25 泛素化和蛋白酶体降解后，参与了抗病毒 RIG-I 样受体激活后 I 型干扰素的产生

Mitochondrial dynamics regulate the RIG-I-like receptor antiviral pathway

线粒体动力学调控RIG-I样受体抗病毒通路

Unpaired 5' ppp-nucleotides, as found in arenavirus double-stranded RNA panhandles, are not recognized by RIG-I

未配对的5' ppp核苷酸（例如沙粒病毒双链RNA的柄状结构中存在的核苷酸）不能被RIG-I识别。

Activation of the beta interferon promoter by unnatural Sendai virus infection requires RIG-I and is inhibited by viral C proteins.

非天然仙台病毒感染激活β干扰素启动子需要RIG-I，并受到病毒C蛋白的抑制

A short peptide at the amino terminus of the Sendai virus C protein acts as an independent element that induces STAT1 instability.

仙台病毒 C 蛋白氨基末端的短肽作为独立元件，可诱导 STAT1 不稳定

The amino-terminal extensions of the longer Sendai virus C proteins modulate pY701-Stat1 and bulk Stat1 levels independently of interferon signaling.

较长的仙台病毒 C 蛋白的氨基末端延伸部分可以独立于干扰素信号传导来调节 pY701-Stat1 和整体 Stat1 的水平

Sendai virus trailer RNA binds TIAR, a cellular protein involved in virus-induced apoptosis

仙台病毒尾部RNA与TIAR结合，TIAR是一种参与病毒诱导细胞凋亡的细胞蛋白