Garvy Beth A相关文献与解析，生知库 | 链接生命科学的每一步

Myeloid arginase-1 controls excessive inflammation and modulates T cell responses in Pseudomonas aeruginosa pneumonia

髓系精氨酸酶-1控制过度炎症并调节铜绿假单胞菌肺炎中的T细胞反应

doi:10.1016/j.imbio.2020.152034

Haydar, Dalia; Gonzalez, Rene; Garvy, Beth A; Garneau-Tsodikova, Sylvie; Thamban Chandrika, Nishad; Bocklage, Therese J; Feola, David J

Platelets Endocytose Viral Particles and Are Activated via TLR (Toll-Like Receptor) Signaling

血小板内吞病毒颗粒并通过TLR（Toll样受体）信号通路被激活

期刊:Arteriosclerosis Thrombosis and Vascular Biology

影响因子:7.4

doi:10.1161/ATVBAHA.120.314180

Banerjee, Meenakshi; Huang, Yunjie; Joshi, Smita; Popa, Gabriel J; Mendenhall, Michael D; Wang, Qing Jun; Garvy, Beth A; Myint, Thein; Whiteheart, Sidney W

The Relationship Between Latent Tuberculosis Infection and Acute Myocardial Infarction

潜伏性结核感染与急性心肌梗死的关系

期刊:Clinical Infectious Diseases

影响因子:7.3

doi:10.1093/cid/cix910

Huaman, Moises A; Ticona, Eduardo; Miranda, Gustavo; Kryscio, Richard J; Mugruza, Raquel; Aranda, Ernesto; Rondan, Paola L; Henson, David; Ticona, Cesar; Sterling, Timothy R; Fichtenbaum, Carl J; Garvy, Beth A

Latent tuberculosis infection is associated with increased unstimulated levels of interferon-gamma in Lima, Peru

在秘鲁利马，潜伏性结核感染与未刺激状态下干扰素-γ水平升高有关。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0202191

Huaman, Moises A; Henson, David; Rondan, Paola L; Ticona, Eduardo; Miranda, Gustavo; Kryscio, Richard J; Mugruza, Raquel; Aranda, Ernesto; Ticona, Cesar; Abarca, Susan; Heredia, Paula; Aguirre, Andres; Sterling, Timothy R; Garvy, Beth A; Fichtenbaum, Carl J

炎症/感染

发表日期：2018

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The trophic life cycle stage of Pneumocystis species induces protective adaptive responses without inflammation-mediated progression to pneumonia

肺孢子虫属的营养期生活史可诱导保护性适应性反应，而不会通过炎症介导发展为肺炎。

期刊:Medical Mycology

影响因子:2.3

doi:10.1093/mmy/myx145

Evans, Heather M; Garvy, Beth A

Overcoming Hurdles to Development of a Vaccine against Pneumocystis jirovecii

克服卡氏肺囊虫肺炎疫苗研发中的障碍

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00035-17

The Trophic Life Cycle Stage of the Opportunistic Fungal Pathogen Pneumocystis murina Hinders the Ability of Dendritic Cells To Stimulate CD4(+) T Cell Responses.

机会性真菌病原体鼠肺孢子虫的营养生命周期阶段会阻碍树突状细胞刺激 CD4(+) T 细胞反应的能力

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00396-17

Evans Heather M, Simpson Andrew, Shen Shu, Stromberg Arnold J, Pickett Carol L, Garvy Beth A

Pneumocystis infection alters the activation state of pulmonary macrophages

肺孢子虫感染会改变肺巨噬细胞的活化状态

期刊:Immunobiology

影响因子:2.3

doi:10.1016/j.imbio.2016.10.003

Deckman, Jessica M; Kurkjian, Cathryn J; McGillis, Joseph P; Cory, Theodore J; Birket, Susan E; Schutzman, Linda M; Murphy, Brian S; Garvy, Beth A; Feola, David J

The life cycle stages of Pneumocystis murina have opposing effects on the immune response to this opportunistic, fungal pathogen

鼠肺孢子虫的生命周期阶段对这种机会性真菌病原体的免疫反应具有相反的影响。

期刊:Infection and Immunity

影响因子:2.8

doi:10.1128/IAI.00519-16

Evans, Heather M; Bryant, Grady L 3rd; Garvy, Beth A

Total Plasma Protein S Is a Prothrombotic Marker in People Living With HIV

总血浆蛋白S是HIV感染者体内的促血栓形成标志物。

Myeloid arginase-1 controls excessive inflammation and modulates T cell responses in Pseudomonas aeruginosa pneumonia

髓系精氨酸酶-1控制过度炎症并调节铜绿假单胞菌肺炎中的T细胞反应

Platelets Endocytose Viral Particles and Are Activated via TLR (Toll-Like Receptor) Signaling

血小板内吞病毒颗粒并通过TLR（Toll样受体）信号通路被激活

The Relationship Between Latent Tuberculosis Infection and Acute Myocardial Infarction

潜伏性结核感染与急性心肌梗死的关系

Latent tuberculosis infection is associated with increased unstimulated levels of interferon-gamma in Lima, Peru

在秘鲁利马，潜伏性结核感染与未刺激状态下干扰素-γ水平升高有关。

The trophic life cycle stage of Pneumocystis species induces protective adaptive responses without inflammation-mediated progression to pneumonia

肺孢子虫属的营养期生活史可诱导保护性适应性反应，而不会通过炎症介导发展为肺炎。

Overcoming Hurdles to Development of a Vaccine against Pneumocystis jirovecii

克服卡氏肺囊虫肺炎疫苗研发中的障碍

The Trophic Life Cycle Stage of the Opportunistic Fungal Pathogen Pneumocystis murina Hinders the Ability of Dendritic Cells To Stimulate CD4(+) T Cell Responses.

机会性真菌病原体鼠肺孢子虫的营养生命周期阶段会阻碍树突状细胞刺激 CD4(+) T 细胞反应的能力

Pneumocystis infection alters the activation state of pulmonary macrophages

肺孢子虫感染会改变肺巨噬细胞的活化状态

The life cycle stages of Pneumocystis murina have opposing effects on the immune response to this opportunistic, fungal pathogen

鼠肺孢子虫的生命周期阶段对这种机会性真菌病原体的免疫反应具有相反的影响。