Gasiewicz相关文献与解析，生知库 | 链接生命科学的每一步

Human and mouse microarrays-guided expression analysis of membrane protein trafficking-related genes in MDCK cells, a canine epithelial model for apical and basolateral differential protein targeting

人类和小鼠微阵列引导的膜蛋白运输相关基因在 MDCK 细胞中的表达分析，MDCK 细胞是一种用于顶端和基底外侧差异蛋白靶向的犬上皮模型

期刊:

影响因子:

doi:10.1016/j.biopen.2017.04.002

Xiaofan Xu, Mingming Pan, Alexis E Gasiewicz, Rongzi Li, Shiu-Ming Kuo

Aryl Hydrocarbon Receptor Deficiency in an Exon 3 Deletion Mouse Model Promotes Hematopoietic Stem Cell Proliferation and Impacts Endosteal Niche Cells

外显子 3 缺失小鼠模型中的芳基烃受体缺陷会促进造血干细胞增殖并影响骨内膜微环境细胞

期刊:Stem Cells International

影响因子:3.8

doi:10.1155/2016/4536187

Zeenath Unnisa, Kameshwar P Singh, Ellen C Henry, Catherine L Donegan, John A Bennett, Thomas A Gasiewicz

Deficiency in Aryl Hydrocarbon Receptor (AHR) Expression throughout Aging Alters Gene Expression Profiles in Murine Long-Term Hematopoietic Stem Cells

衰老过程中芳基烃受体 (AHR) 表达的缺乏会改变小鼠长期造血干细胞的基因表达谱

期刊:PLoS One

影响因子:2.9

doi:10.1371/journal.pone.0133791

John A Bennett, Kameshwar P Singh, Zeenath Unnisa, Stephen L Welle, Thomas A Gasiewicz

The heat shock protein 90 inhibitor, (-)-epigallocatechin gallate, has anticancer activity in a novel human prostate cancer progression model

热休克蛋白90抑制剂(-)-表没食子儿茶素没食子酸酯在一种新型人类前列腺癌进展模型中具有抗癌活性。

期刊:Cancer Prevention Research

影响因子:2.6

doi:10.1158/1940-6207.CAPR-14-0224

Moses, Michael A; Henry, Ellen C; Ricke, William A; Gasiewicz, Thomas A

Loss of aryl hydrocarbon receptor promotes gene changes associated with premature hematopoietic stem cell exhaustion and development of a myeloproliferative disorder in aging mice

芳烃受体的丧失会促进与衰老小鼠过早造血干细胞衰竭和骨髓增生性疾病发展相关的基因变化

期刊:Stem Cells and Development

影响因子:2.5

doi:10.1089/scd.2013.0346

Kameshwar P Singh, John A Bennett, Fanny L Casado, Jason L Walrath, Stephen L Welle, Thomas A Gasiewicz

Aryl hydrocarbon receptor activation in hematopoietic stem/progenitor cells alters cell function and pathway-specific gene modulation reflecting changes in cellular trafficking and migration

造血干细胞/祖细胞中的芳烃受体激活会改变细胞功能和通路特异性基因调节，从而反映细胞运输和迁移的变化

期刊:Molecular Pharmacology

影响因子:3.2

doi:10.1124/mol.111.071381

Fanny L Casado, Kameshwar P Singh, Thomas A Gasiewicz

Aryl hydrocarbon receptor-null allele mice have hematopoietic stem/progenitor cells with abnormal characteristics and functions

芳烃受体基因敲除小鼠的造血干/祖细胞具有异常的特征和功能。

期刊:

影响因子:

doi:10.1089/scd.2010.0333

Singh, Kameshwar P; Garrett, Russell W; Casado, Fanny L; Gasiewicz, Thomas A

Neural precursor cell proliferation is disrupted through activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxin

2,3,7,8-四氯二苯并二恶英通过激活芳烃受体来抑制神经前体细胞增殖。

期刊:

影响因子:

doi:10.1089/scd.2009.0529

Latchney, Sarah E; Lioy, Daniel T; Henry, Ellen C; Gasiewicz, Thomas A; Strathmann, Frederick G; Mayer-Pröschel, Margot; Opanashuk, Lisa A

Conditional deletion of Ahr alters gene expression profiles in hematopoietic stem cells

条件性删除 Ahr 会改变造血干细胞的基因表达谱

Aryl Hydrocarbon Receptor and Stem Cells

芳烃受体和干细胞

Human and mouse microarrays-guided expression analysis of membrane protein trafficking-related genes in MDCK cells, a canine epithelial model for apical and basolateral differential protein targeting

人类和小鼠微阵列引导的膜蛋白运输相关基因在 MDCK 细胞中的表达分析，MDCK 细胞是一种用于顶端和基底外侧差异蛋白靶向的犬上皮模型

Aryl Hydrocarbon Receptor Deficiency in an Exon 3 Deletion Mouse Model Promotes Hematopoietic Stem Cell Proliferation and Impacts Endosteal Niche Cells

外显子 3 缺失小鼠模型中的芳基烃受体缺陷会促进造血干细胞增殖并影响骨内膜微环境细胞

Deficiency in Aryl Hydrocarbon Receptor (AHR) Expression throughout Aging Alters Gene Expression Profiles in Murine Long-Term Hematopoietic Stem Cells

衰老过程中芳基烃受体 (AHR) 表达的缺乏会改变小鼠长期造血干细胞的基因表达谱

The heat shock protein 90 inhibitor, (-)-epigallocatechin gallate, has anticancer activity in a novel human prostate cancer progression model

热休克蛋白90抑制剂(-)-表没食子儿茶素没食子酸酯在一种新型人类前列腺癌进展模型中具有抗癌活性。

Loss of aryl hydrocarbon receptor promotes gene changes associated with premature hematopoietic stem cell exhaustion and development of a myeloproliferative disorder in aging mice

芳烃受体的丧失会促进与衰老小鼠过早造血干细胞衰竭和骨髓增生性疾病发展相关的基因变化

Aryl hydrocarbon receptor activation in hematopoietic stem/progenitor cells alters cell function and pathway-specific gene modulation reflecting changes in cellular trafficking and migration

造血干细胞/祖细胞中的芳烃受体激活会改变细胞功能和通路特异性基因调节，从而反映细胞运输和迁移的变化

Aryl hydrocarbon receptor-null allele mice have hematopoietic stem/progenitor cells with abnormal characteristics and functions

芳烃受体基因敲除小鼠的造血干/祖细胞具有异常的特征和功能。

Neural precursor cell proliferation is disrupted through activation of the aryl hydrocarbon receptor by 2,3,7,8-tetrachlorodibenzo-p-dioxin

2,3,7,8-四氯二苯并二恶英通过激活芳烃受体来抑制神经前体细胞增殖。