Gatt Anthony相关文献与解析，生知库 | 链接生命科学的每一步

Comparison of Heterologous Prime-Boost Strategies against Human Immunodeficiency Virus Type 1 Gag Using Negative Stranded RNA Viruses

利用负链RNA病毒比较针对人类免疫缺陷病毒1型Gag的异源初免-加强免疫策略

doi:10.1371/journal.pone.0067123

Lawrence, Tessa M; Wanjalla, Celestine N; Gomme, Emily A; Wirblich, Christoph; Gatt, Anthony; Carnero, Elena; García-Sastre, Adolfo; Lyles, Douglas S; McGettigan, James P; Schnell, Matthias J

Protective vaccine-induced CD4(+) T cell-independent B cell responses against rabies infection

疫苗诱导的针对狂犬病感染的CD4(+) T细胞非依赖性B细胞保护性反应

期刊:Journal of Virology

影响因子:3.8

doi:10.1128/JVI.00615-12

Dorfmeier, Corin L; Lytle, Andrew G; Dunkel, Amber L; Gatt, Anthony; McGettigan, James P

Tau Ser262 phosphorylation is critical for Abeta42-induced tau toxicity in a transgenic Drosophila model of Alzheimer's disease

在阿尔茨海默病转基因果蝇模型中，Tau Ser262磷酸化对于Aβ42诱导的tau蛋白毒性至关重要。

期刊:Human Molecular Genetics

影响因子:3.2

doi:10.1093/hmg/ddq200

Iijima, Koichi; Gatt, Anthony; Iijima-Ando, Kanae

A DNA damage-activated checkpoint kinase phosphorylates tau and enhances tau-induced neurodegeneration.

DNA损伤激活的检查点激酶可磷酸化tau蛋白，并增强tau蛋白诱导的神经退行性变

期刊:Human Molecular Genetics

影响因子:3.2

doi:10.1093/hmg/ddq068

Iijima-Ando Kanae, Zhao LiJuan, Gatt Anthony, Shenton Christopher, Iijima Koichi

Mitochondrial mislocalization underlies Abeta42-induced neuronal dysfunction in a Drosophila model of Alzheimer's disease

在果蝇阿尔茨海默病模型中，线粒体错位是Aβ42诱导神经元功能障碍的根本原因。

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0008310

Iijima-Ando, Kanae; Hearn, Stephen A; Shenton, Christopher; Gatt, Anthony; Zhao, Lijuan; Iijima, Koichi

Overexpression of neprilysin reduces alzheimer amyloid-beta42 (Abeta42)-induced neuron loss and intraneuronal Abeta42 deposits but causes a reduction in cAMP-responsive element-binding protein-mediated transcription, age-dependent axon pathology, and premature death in Drosophila

脑啡肽酶的过表达可减少阿尔茨海默病淀粉样蛋白β42 (Aβ42) 诱导的神经元丢失和神经元内Aβ42沉积，但会导致cAMP反应元件结合蛋白介导的转录减少、年龄依赖性轴突病变以及果蝇过早死亡。

期刊:Journal of Biological Chemistry

影响因子:3.9

doi:10.1074/jbc.M710509200

Iijima-Ando, Kanae; Hearn, Stephen A; Granger, Linda; Shenton, Christopher; Gatt, Anthony; Chiang, Hsueh-Cheng; Hakker, Inessa; Zhong, Yi; Iijima, Koichi

Abeta42 mutants with different aggregation profiles induce distinct pathologies in Drosophila

具有不同聚集特征的Aβ42突变体在果蝇中诱发不同的病理

期刊:PLoS One

影响因子:2.6

doi:10.1371/journal.pone.0001703

Iijima, Koichi; Chiang, Hsueh-Cheng; Hearn, Stephen A; Hakker, Inessa; Gatt, Anthony; Shenton, Christopher; Granger, Linda; Leung, Amy; Iijima-Ando, Kanae; Zhong, Yi

Drosophila

发表日期：2008

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Ethical considerations in the regulation and use of herbal medicines in the European Union

欧盟草药监管和使用中的伦理考量

Comparison of Heterologous Prime-Boost Strategies against Human Immunodeficiency Virus Type 1 Gag Using Negative Stranded RNA Viruses

利用负链RNA病毒比较针对人类免疫缺陷病毒1型Gag的异源初免-加强免疫策略

Protective vaccine-induced CD4(+) T cell-independent B cell responses against rabies infection

疫苗诱导的针对狂犬病感染的CD4(+) T细胞非依赖性B细胞保护性反应

Tau Ser262 phosphorylation is critical for Abeta42-induced tau toxicity in a transgenic Drosophila model of Alzheimer's disease

在阿尔茨海默病转基因果蝇模型中，Tau Ser262磷酸化对于Aβ42诱导的tau蛋白毒性至关重要。

A DNA damage-activated checkpoint kinase phosphorylates tau and enhances tau-induced neurodegeneration.

DNA损伤激活的检查点激酶可磷酸化tau蛋白，并增强tau蛋白诱导的神经退行性变

Mitochondrial mislocalization underlies Abeta42-induced neuronal dysfunction in a Drosophila model of Alzheimer's disease

在果蝇阿尔茨海默病模型中，线粒体错位是Aβ42诱导神经元功能障碍的根本原因。

Overexpression of neprilysin reduces alzheimer amyloid-beta42 (Abeta42)-induced neuron loss and intraneuronal Abeta42 deposits but causes a reduction in cAMP-responsive element-binding protein-mediated transcription, age-dependent axon pathology, and premature death in Drosophila

脑啡肽酶的过表达可减少阿尔茨海默病淀粉样蛋白β42 (Aβ42) 诱导的神经元丢失和神经元内Aβ42沉积，但会导致cAMP反应元件结合蛋白介导的转录减少、年龄依赖性轴突病变以及果蝇过早死亡。

Abeta42 mutants with different aggregation profiles induce distinct pathologies in Drosophila

具有不同聚集特征的Aβ42突变体在果蝇中诱发不同的病理